FKBP38 protects Bcl-2 from caspase-dependent degradation

• المؤلفون الرئيسيون: Choi, Bo-Hwa, Feng, Lin, Yoon, Ho Sup
• مؤلفون آخرون: School of Biological Sciences
• التنسيق: مقال
• اللغة: English
• منشور في: 2012
• الموضوعات: DRNTU::Science::Biological sciences
• الوصول للمادة أونلاين: https://hdl.handle.net/10356/95127; http://hdl.handle.net/10220/8819

The cellular processes that regulate Bcl-2 at the posttranslational levels are as important as those that regulate bcl-2 synthesis. Previously we demonstrated that the suppression of FK506-binding protein 38 (FKBP38) contributes to the instability of Bcl-2 or leaves Bcl-2 unprotected from degradatio...