Influenza A virus facilitates its infectivity by activating p53 to inhibit the expression of interferon-induced transmembrane proteins

• المؤلفون الرئيسيون: Wang, B., Lam, T.H., Soh, M.K., Ye, Z., Chen, J., Ren, E.C.
• مؤلفون آخرون: MICROBIOLOGY AND IMMUNOLOGY
• التنسيق: مقال
• منشور في: Frontiers Media S.A. 2020
• الموضوعات: antinuclear antibody; beta interferon; caspase 3; caspase 7; fluorouracil; interferon; membrane protein; nutlin 3; protein MDM2; protein p53; small interfering RNA; apoptosis; Article; cell viability assay; chemiluminescence immunoassay; controlled study; CRISPR-CAS9 system; cytotoxicity; endosome; epithelium cell; gene expression; gene silencing; genetic transfection; human; human cell; immunofluorescence test; in vitro propagation; Influenza A virus; innate immunity; microarray analysis; plasmid; protein expression; real time polymerase chain reaction; receptor down regulation; receptor upregulation; respiratory tract infection; RNA isolation; sequence analysis; technology; transcription regulation; virus infectivity; virus replication; Western blotting
• الوصول للمادة أونلاين: https://scholarbank.nus.edu.sg/handle/10635/174536
• المؤسسة: National University of Singapore

10.3389/fimmu.2018.01193