Adipocyte ALK7 links nutrient overload to catecholamine resistance in obesity

• المؤلفون الرئيسيون: Guo, T, Marmol, P, Moliner, A, Bj”rnholm, M, Zhang, C, Shokat, K.M, Ibanez, C.F
• مؤلفون آخرون: PHYSIOLOGY
• التنسيق: مقال
• منشور في: 2020
• الموضوعات: 1-tert-butyl-3-naphthalen-1-ylmethyl-1H-pyrazolo(3,4-d)pyrimidin-4-ylemine; activin receptor 1; Acvr1c protein, mouse; adenosine triphosphate; beta adrenergic receptor; catecholamine; fat intake; pyrazole derivative; pyrimidine derivative; transforming growth factor beta; adipocyte; adipose tissue; adverse effects; animal; antagonists and inhibitors; deficiency; drug effects; gene expression regulation; genetics; human; knockout mouse; lipid diet; lipid peroxidation; lipolysis; metabolism; mitochondrial dynamics; mouse; obesity; pathology; primary cell culture; signal transduction; Activin Receptors, Type I; Adenosine Triphosphate; Adipocytes; Adipose Tissue; Animals; Catecholamines; Diet, High-Fat; Dietary Fats; Gene Expression Regulation; Humans; Lipid Peroxidation; Lipolysis; Mice; Mice, Knockout; Mitochondrial Turnover; Obesity; Primary Cell Culture; Pyrazoles; Pyrimidines; Receptors, Adrenergic, beta; Signal Transduction; Transforming Growth Factor beta
• الوصول للمادة أونلاين: https://scholarbank.nus.edu.sg/handle/10635/176018

10.7554/eLife.03245