Glucose activates the carboxyl methylation of subunits of trimeric GTP- binding proteins in pancreatic β cells: Modulation in vivo by calcium, GTP, and pertussis toxin

Journal of Clinical Investigation

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Main Authors:	Kowluru, A., Li, G., Metz, S.A.
Other Authors:	NATIONAL UNIVERSITY MEDICAL INSTITUTES
Format:	Article
Published:	2014
Subjects:	Cyclooxygenase inhibitors and insulin secretion GTP-binding proteins Pancreatic β cells Prostaglandin E2 Subunit carboxyl methylation
Online Access:	http://scholarbank.nus.edu.sg/handle/10635/112792
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Institution:	National University of Singapore

id	sg-nus-scholar.10635-112792
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spelling	sg-nus-scholar.10635-1127922015-02-15T08:10:00Z Glucose activates the carboxyl methylation of subunits of trimeric GTP- binding proteins in pancreatic β cells: Modulation in vivo by calcium, GTP, and pertussis toxin Kowluru, A. Li, G. Metz, S.A. NATIONAL UNIVERSITY MEDICAL INSTITUTES Cyclooxygenase inhibitors and insulin secretion GTP-binding proteins Pancreatic β cells Prostaglandin E2 Subunit carboxyl methylation Journal of Clinical Investigation 100 6 1596-1610 JCINA 2014-11-28T07:56:51Z 2014-11-28T07:56:51Z 1997-09-15 Article Kowluru, A.,Li, G.,Metz, S.A. (1997-09-15). Glucose activates the carboxyl methylation of subunits of trimeric GTP- binding proteins in pancreatic β cells: Modulation in vivo by calcium, GTP, and pertussis toxin. Journal of Clinical Investigation 100 (6) : 1596-1610. ScholarBank@NUS Repository. 00219738 http://scholarbank.nus.edu.sg/handle/10635/112792 NOT_IN_WOS Scopus
institution	National University of Singapore
building	NUS Library
country	Singapore
collection	ScholarBank@NUS
topic	Cyclooxygenase inhibitors and insulin secretion GTP-binding proteins Pancreatic β cells Prostaglandin E2 Subunit carboxyl methylation
spellingShingle	Cyclooxygenase inhibitors and insulin secretion GTP-binding proteins Pancreatic β cells Prostaglandin E2 Subunit carboxyl methylation Kowluru, A. Li, G. Metz, S.A. Glucose activates the carboxyl methylation of subunits of trimeric GTP- binding proteins in pancreatic β cells: Modulation in vivo by calcium, GTP, and pertussis toxin
description	Journal of Clinical Investigation
author2	NATIONAL UNIVERSITY MEDICAL INSTITUTES
author_facet	NATIONAL UNIVERSITY MEDICAL INSTITUTES Kowluru, A. Li, G. Metz, S.A.
format	Article
author	Kowluru, A. Li, G. Metz, S.A.
author_sort	Kowluru, A.
title	Glucose activates the carboxyl methylation of subunits of trimeric GTP- binding proteins in pancreatic β cells: Modulation in vivo by calcium, GTP, and pertussis toxin
title_short	Glucose activates the carboxyl methylation of subunits of trimeric GTP- binding proteins in pancreatic β cells: Modulation in vivo by calcium, GTP, and pertussis toxin
title_full	Glucose activates the carboxyl methylation of subunits of trimeric GTP- binding proteins in pancreatic β cells: Modulation in vivo by calcium, GTP, and pertussis toxin
title_fullStr	Glucose activates the carboxyl methylation of subunits of trimeric GTP- binding proteins in pancreatic β cells: Modulation in vivo by calcium, GTP, and pertussis toxin
title_full_unstemmed	Glucose activates the carboxyl methylation of subunits of trimeric GTP- binding proteins in pancreatic β cells: Modulation in vivo by calcium, GTP, and pertussis toxin
title_sort	glucose activates the carboxyl methylation of subunits of trimeric gtp- binding proteins in pancreatic β cells: modulation in vivo by calcium, gtp, and pertussis toxin
publishDate	2014
url	http://scholarbank.nus.edu.sg/handle/10635/112792
_version_	1681094548139278336

Glucose activates the carboxyl methylation of subunits of trimeric GTP- binding proteins in pancreatic β cells: Modulation in vivo by calcium, GTP, and pertussis toxin

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