Mutations in CCDC11, which encodes a coiled-coil containing ciliary protein, causes situs inversus due to dysmotility of monocilia in the left-right organizer

10.1002/humu.22738

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Main Authors: Narasimhan, Vijayashankaranarayanan, Hjeij, Rim, Vij, Shubha, Loges, Niki Tomas, Wallmeier, Julia, Koerner-Rettberg, Cordula, Werner, Claudius, Thamilselvam, Surin Kumar, Boey, Adrian, Choksi, Semil P., Pennekamp, Petra, Roy, Sudipto, Omran, Heymut
Other Authors: BIOLOGICAL SCIENCES
Format: Article
Published: Wiley 2015
Online Access:http://scholarbank.nus.edu.sg/handle/10635/119970
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spelling sg-nus-scholar.10635-1199702023-10-25T21:27:39Z Mutations in CCDC11, which encodes a coiled-coil containing ciliary protein, causes situs inversus due to dysmotility of monocilia in the left-right organizer Narasimhan, Vijayashankaranarayanan Hjeij, Rim Vij, Shubha Loges, Niki Tomas Wallmeier, Julia Koerner-Rettberg, Cordula Werner, Claudius Thamilselvam, Surin Kumar Boey, Adrian Choksi, Semil P. Pennekamp, Petra Roy, Sudipto Omran, Heymut BIOLOGICAL SCIENCES 10.1002/humu.22738 Human Mutation  36 3 307-318 2015-06-22T07:56:01Z 2015-06-22T07:56:01Z 2015-03-01 Article Narasimhan, Vijayashankaranarayanan, Hjeij, Rim, Vij, Shubha, Loges, Niki Tomas, Wallmeier, Julia, Koerner-Rettberg, Cordula, Werner, Claudius, Thamilselvam, Surin Kumar, Boey, Adrian, Choksi, Semil P., Pennekamp, Petra, Roy, Sudipto, Omran, Heymut (2015-03-01). Mutations in CCDC11, which encodes a coiled-coil containing ciliary protein, causes situs inversus due to dysmotility of monocilia in the left-right organizer. Human Mutation  36 (3) : 307-318. ScholarBank@NUS Repository. https://doi.org/10.1002/humu.22738 10597794 15308561 http://scholarbank.nus.edu.sg/handle/10635/119970 000351399900006 Wiley
institution National University of Singapore
building NUS Library
continent Asia
country Singapore
Singapore
content_provider NUS Library
collection ScholarBank@NUS
description 10.1002/humu.22738
author2 BIOLOGICAL SCIENCES
author_facet BIOLOGICAL SCIENCES
Narasimhan, Vijayashankaranarayanan
Hjeij, Rim
Vij, Shubha
Loges, Niki Tomas
Wallmeier, Julia
Koerner-Rettberg, Cordula
Werner, Claudius
Thamilselvam, Surin Kumar
Boey, Adrian
Choksi, Semil P.
Pennekamp, Petra
Roy, Sudipto
Omran, Heymut
format Article
author Narasimhan, Vijayashankaranarayanan
Hjeij, Rim
Vij, Shubha
Loges, Niki Tomas
Wallmeier, Julia
Koerner-Rettberg, Cordula
Werner, Claudius
Thamilselvam, Surin Kumar
Boey, Adrian
Choksi, Semil P.
Pennekamp, Petra
Roy, Sudipto
Omran, Heymut
spellingShingle Narasimhan, Vijayashankaranarayanan
Hjeij, Rim
Vij, Shubha
Loges, Niki Tomas
Wallmeier, Julia
Koerner-Rettberg, Cordula
Werner, Claudius
Thamilselvam, Surin Kumar
Boey, Adrian
Choksi, Semil P.
Pennekamp, Petra
Roy, Sudipto
Omran, Heymut
Mutations in CCDC11, which encodes a coiled-coil containing ciliary protein, causes situs inversus due to dysmotility of monocilia in the left-right organizer
author_sort Narasimhan, Vijayashankaranarayanan
title Mutations in CCDC11, which encodes a coiled-coil containing ciliary protein, causes situs inversus due to dysmotility of monocilia in the left-right organizer
title_short Mutations in CCDC11, which encodes a coiled-coil containing ciliary protein, causes situs inversus due to dysmotility of monocilia in the left-right organizer
title_full Mutations in CCDC11, which encodes a coiled-coil containing ciliary protein, causes situs inversus due to dysmotility of monocilia in the left-right organizer
title_fullStr Mutations in CCDC11, which encodes a coiled-coil containing ciliary protein, causes situs inversus due to dysmotility of monocilia in the left-right organizer
title_full_unstemmed Mutations in CCDC11, which encodes a coiled-coil containing ciliary protein, causes situs inversus due to dysmotility of monocilia in the left-right organizer
title_sort mutations in ccdc11, which encodes a coiled-coil containing ciliary protein, causes situs inversus due to dysmotility of monocilia in the left-right organizer
publisher Wiley
publishDate 2015
url http://scholarbank.nus.edu.sg/handle/10635/119970
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