Laminin ?4 deficient mice exhibit decreased capacity for adipose tissue expansion and weight gain

• المؤلفون الرئيسيون: Vaicik M.K., Kortesmaa J.T., Movérare-Skrtic S., Kortesmaa J., Soininen R., Bergström G., Ohlsson C., Chong L.Y., Rozell B., Emont M., Cohen R.N., Brey E.M., Tryggvason K.
• مؤلفون آخرون: NUSHS PROJECT
• التنسيق: مقال
• منشور في: 2019
• الموضوعات: laminin alpha4; Lama4 protein, mouse; laminin; adipose tissue; age; animal cell; animal tissue; Article; bioaccumulation; controlled study; epididymis fat; extracellular matrix; fat mass; knockout mouse; laminin alpha4 gene; lipid composition; lipid diet; lipogenesis; male; mouse; nonhuman; obesity; protein function; tissue expansion; tissue structure; weight gain; wild type; adipocyte; aging; animal; C57BL mouse; cell size; deficiency; feeding behavior; metabolism; pathology; subcutaneous fat; white adipose tissue; Mus; Adipocytes; Adipose Tissue; Adipose Tissue, White; Aging; Animals; Cell Size; Diet, High-Fat; Feeding Behavior; Laminin; Lipogenesis; Male; Mice, Inbred C57BL; Obesity; Subcutaneous Fat; Weight Gain
• الوصول للمادة أونلاين: https://scholarbank.nus.edu.sg/handle/10635/161768

10.1371/journal.pone.0109854