Gene expression profiling and network analysis reveals lipid and steroid metabolism to be the most favored by TNF? in HepG2 cells

• Main Authors: Pandey A.K., Munjal N., Datta M.
• Other Authors: CANCER SCIENCE INSTITUTE OF SINGAPORE
• Format: Article
• Published: 2019
• Subjects: actin related protein; aquaporin 3; CCAAT enhancer binding protein alpha; DDX54 protein; DNA methyltransferase 3B; E47 protein; fat; fibroblast growth factor receptor 5; Freac 7 protein; heat shock protein 90; herpesvirus entry mediator; immunoglobulin E; immunoglobulin enhancer binding protein; immunoglobulin receptor; interferon regulatory factor 1; laminin; lipid; MEF 2 protein; protein; protein p300; Ryk protein; SLC25A5 protein; sterol regulatory element binding protein 1; TBL1X protein; tissue inhibitor of metalloproteinase 1; transcription factor foxd3; transcription factor FOXJ2; transcription factor NF E2; tumor necrosis factor alpha; unclassified drug; unindexed drug; steroid; article; binding site; cancer cell culture; down regulation; gene cluster; gene expression; gene expression profiling; human; human cell; human cell culture; immune response; immunity; lipid metabolism; liver cell; liver cell carcinoma; microarray analysis; nucleotide sequence; protein binding; steroid metabolism; upregulation; cell strain HepG2; cluster analysis; DNA microarray; drug effect; gene expression regulation; gene regulatory network; genetics; metabolism; reverse transcription polymerase chain reaction; Cluster Analysis; Gene Expression Profiling; Gene Expression Regulation, Neoplastic; Gene Regulatory Networks; Hep G2 Cells; Humans; Lipid Metabolism; Oligonucleotide Array Sequence Analysis; Reverse Transcriptase Polymerase Chain Reaction; Steroids; Tumor Necrosis Factor-alpha
• Online Access: https://scholarbank.nus.edu.sg/handle/10635/161818
• Institution: National University of Singapore

10.1371/journal.pone.0009063