The arginase inhibitor N ? -hydroxy-nor-arginine (nor-NOHA) induces apoptosis in leukemic cells specifically under hypoxic conditions but CRISPR/Cas9 excludes arginase 2 (ARG2) as the functional target

10.1371/journal.pone.0205254

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Main Authors:	Ng K.P., Manjeri A., Lee L.M., Chan Z.E., Tan C.Y., Tan Q.D., Majeed A., Lee K.L., Chuah C., Suda T., Ong S.T.
Other Authors:	CANCER SCIENCE INSTITUTE OF SINGAPORE
Format:	Article
Published:	Public Library of Science 2020
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/164158
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Institution:	National University of Singapore

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spelling	sg-nus-scholar.10635-1641582023-10-31T08:23:46Z The arginase inhibitor N ? -hydroxy-nor-arginine (nor-NOHA) induces apoptosis in leukemic cells specifically under hypoxic conditions but CRISPR/Cas9 excludes arginase 2 (ARG2) as the functional target Ng K.P. Manjeri A. Lee L.M. Chan Z.E. Tan C.Y. Tan Q.D. Majeed A. Lee K.L. Chuah C. Suda T. Ong S.T. CANCER SCIENCE INSTITUTE OF SINGAPORE DEPT OF MEDICINE DUKE-NUS MEDICAL SCHOOL 10.1371/journal.pone.0205254 PLoS ONE 13 10 e0205254 2020-01-31T10:43:43Z 2020-01-31T10:43:43Z 2018 Article Ng K.P., Manjeri A., Lee L.M., Chan Z.E., Tan C.Y., Tan Q.D., Majeed A., Lee K.L., Chuah C., Suda T., Ong S.T. (2018). The arginase inhibitor N ? -hydroxy-nor-arginine (nor-NOHA) induces apoptosis in leukemic cells specifically under hypoxic conditions but CRISPR/Cas9 excludes arginase 2 (ARG2) as the functional target. PLoS ONE 13 (10) : e0205254. ScholarBank@NUS Repository. https://doi.org/10.1371/journal.pone.0205254 19326203 https://scholarbank.nus.edu.sg/handle/10635/164158 Public Library of Science
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
description	10.1371/journal.pone.0205254
author2	CANCER SCIENCE INSTITUTE OF SINGAPORE
author_facet	CANCER SCIENCE INSTITUTE OF SINGAPORE Ng K.P. Manjeri A. Lee L.M. Chan Z.E. Tan C.Y. Tan Q.D. Majeed A. Lee K.L. Chuah C. Suda T. Ong S.T.
format	Article
author	Ng K.P. Manjeri A. Lee L.M. Chan Z.E. Tan C.Y. Tan Q.D. Majeed A. Lee K.L. Chuah C. Suda T. Ong S.T.
spellingShingle	Ng K.P. Manjeri A. Lee L.M. Chan Z.E. Tan C.Y. Tan Q.D. Majeed A. Lee K.L. Chuah C. Suda T. Ong S.T. The arginase inhibitor N ? -hydroxy-nor-arginine (nor-NOHA) induces apoptosis in leukemic cells specifically under hypoxic conditions but CRISPR/Cas9 excludes arginase 2 (ARG2) as the functional target
author_sort	Ng K.P.
title	The arginase inhibitor N ? -hydroxy-nor-arginine (nor-NOHA) induces apoptosis in leukemic cells specifically under hypoxic conditions but CRISPR/Cas9 excludes arginase 2 (ARG2) as the functional target
title_short	The arginase inhibitor N ? -hydroxy-nor-arginine (nor-NOHA) induces apoptosis in leukemic cells specifically under hypoxic conditions but CRISPR/Cas9 excludes arginase 2 (ARG2) as the functional target
title_full	The arginase inhibitor N ? -hydroxy-nor-arginine (nor-NOHA) induces apoptosis in leukemic cells specifically under hypoxic conditions but CRISPR/Cas9 excludes arginase 2 (ARG2) as the functional target
title_fullStr	The arginase inhibitor N ? -hydroxy-nor-arginine (nor-NOHA) induces apoptosis in leukemic cells specifically under hypoxic conditions but CRISPR/Cas9 excludes arginase 2 (ARG2) as the functional target
title_full_unstemmed	The arginase inhibitor N ? -hydroxy-nor-arginine (nor-NOHA) induces apoptosis in leukemic cells specifically under hypoxic conditions but CRISPR/Cas9 excludes arginase 2 (ARG2) as the functional target
title_sort	arginase inhibitor n ? -hydroxy-nor-arginine (nor-noha) induces apoptosis in leukemic cells specifically under hypoxic conditions but crispr/cas9 excludes arginase 2 (arg2) as the functional target
publisher	Public Library of Science
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/164158
_version_	1781791914597548032

The arginase inhibitor N ? -hydroxy-nor-arginine (nor-NOHA) induces apoptosis in leukemic cells specifically under hypoxic conditions but CRISPR/Cas9 excludes arginase 2 (ARG2) as the functional target

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