Semaphorin 3E/PlexinD1 Signaling Is Required for Cardiac Ventricular Compaction.
10.1172/jci.insight.125908
Saved in:
Main Authors: | SANDIREDDY REDDEMMA, DASAN MARY CIBI, Gupta P, ANAMIKA SINGH, Tee N, Uemura A, Epstein JA, Singh Manvendra Kumar |
---|---|
Other Authors: | DUKE-NUS MEDICAL SCHOOL |
Format: | Article |
Published: |
The American Society for Clinical Investigation
2020
|
Online Access: | https://scholarbank.nus.edu.sg/handle/10635/168452 |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Institution: | National University of Singapore |
Similar Items
-
Semaphorin-PlexinD1 Signaling Limits Angiogenic Potential via the VEGF Decoy Receptor sFlt1
by: Zygmunt, Tomasz, et al.
Published: (2021) -
Semaphorin3E-PlexinD1 signaling in coronary artery and lymphatic vessel development with clinical implications in myocardial recovery
by: Maruyama, Kazuaki, et al.
Published: (2021) -
Role of Semaphorin3E-PlexinD1 signaling in endothelial cell fate determination
by: Goh, Joyce Wei Ting
Published: (2016) -
Tie2Cre-mediated inactivation of plexinD1 results in congenital heart, vascular and skeletal defects
by: Zhang, Ying, et al.
Published: (2021) -
Deficiency in the Secreted Protein Semaphorin3d Causes Abnormal Parathyroid Development in Mice.
by: ANAMIKA SINGH, et al.
Published: (2020)