Loss of maternal Trim28 causes male-predominant early embryonic lethality

• Main Authors: Kumar, A.S, Seah, M.K.Y, Ling, K.Y, Wang, Y, Tan, J.H.L, Nitsch, S, Lim, S.L, Lorthongpanich, C, Wollmann, H, Low, D.H.P, Guccione, E, Messerschmidt, D.M
• Other Authors: BIOCHEMISTRY
• Format: Article
• Published: Cold Spring Harbor Laboratory Press 2020
• Subjects: RNA; tripartite motif protein; tripartite motif protein 28; unclassified drug; nuclear protein; Rbmy1a1 protein, mouse; repressor protein; RNA binding protein; Trim28 protein, mouse; animal cell; animal tissue; Article; blastocyst; comparative study; controlled study; decidualization; demethylation; DNA methylation; embryo; epigenetics; female; fetus death; gene; gene expression regulation; lethality; male; mouse; nonhuman; nuclear reprogramming; phenotype; preimplantation embryo; priority journal; promoter region; Rbmy1a1 gene; RNA sequence; sex chromosome; sperm; X chromosome; Y chromosomal inheritance; animal; cell culture; embryo development; genetic epigenesis; genetics; genome imprinting; mammalian embryo; metabolism; mutation; nidation; spermatozoon; Animals; Cells, Cultured; Cellular Reprogramming; DNA Methylation; Embryo Implantation; Embryo, Mammalian; Embryonic Development; Epigenesis, Genetic; Female; Gene Expression Regulation, Developmental; Genomic Imprinting; Male; Mutation; Nuclear Proteins; Promoter Regions, Genetic; Repressor Proteins; RNA-Binding Proteins; Spermatozoa
• Online Access: https://scholarbank.nus.edu.sg/handle/10635/175239
• Institution: National University of Singapore

10.1101/gad.291195.116