Crosstalk between Bcl-2 family and Ras family small GTPases: potential cell fate regulation?
10.3389/fonc.2012.00206
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sg-nus-scholar.10635-1761792024-04-04T01:08:53Z Crosstalk between Bcl-2 family and Ras family small GTPases: potential cell fate regulation? Kang, J Pervaiz, S PHYSIOLOGY ARF protein cisplatin doxorubicin etoposide gefitinib guanosine triphosphatase hypoxia inducible factor 1 imatinib mevinolin mitogen activated protein kinase paclitaxel protein bcl 2 protein bcl xl protein kinase C Rab protein Rac protein Rac1 protein Raf protein Ran protein Rap protein Ras protein reactive oxygen metabolite Rheb protein Rho kinase stress activated protein kinase sulindac acute lymphoblastic leukemia apoptosis breast cancer cell death cell fate cell permeabilization cell proliferation cellular distribution down regulation drug inactivation gene overexpression human lung non small cell cancer ovary cancer prostate cancer protein processing protein synthesis regulation review signal transduction ubiquitination 10.3389/fonc.2012.00206 Frontiers in Oncology 43467 Article 206 2020-09-14T08:23:30Z 2020-09-14T08:23:30Z 2013 Review Kang, J, Pervaiz, S (2013). Crosstalk between Bcl-2 family and Ras family small GTPases: potential cell fate regulation?. Frontiers in Oncology 43467 : Article 206. ScholarBank@NUS Repository. https://doi.org/10.3389/fonc.2012.00206 2234-943X https://scholarbank.nus.edu.sg/handle/10635/176179 Unpaywall 20200831 |
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ARF protein cisplatin doxorubicin etoposide gefitinib guanosine triphosphatase hypoxia inducible factor 1 imatinib mevinolin mitogen activated protein kinase paclitaxel protein bcl 2 protein bcl xl protein kinase C Rab protein Rac protein Rac1 protein Raf protein Ran protein Rap protein Ras protein reactive oxygen metabolite Rheb protein Rho kinase stress activated protein kinase sulindac acute lymphoblastic leukemia apoptosis breast cancer cell death cell fate cell permeabilization cell proliferation cellular distribution down regulation drug inactivation gene overexpression human lung non small cell cancer ovary cancer prostate cancer protein processing protein synthesis regulation review signal transduction ubiquitination |
spellingShingle |
ARF protein cisplatin doxorubicin etoposide gefitinib guanosine triphosphatase hypoxia inducible factor 1 imatinib mevinolin mitogen activated protein kinase paclitaxel protein bcl 2 protein bcl xl protein kinase C Rab protein Rac protein Rac1 protein Raf protein Ran protein Rap protein Ras protein reactive oxygen metabolite Rheb protein Rho kinase stress activated protein kinase sulindac acute lymphoblastic leukemia apoptosis breast cancer cell death cell fate cell permeabilization cell proliferation cellular distribution down regulation drug inactivation gene overexpression human lung non small cell cancer ovary cancer prostate cancer protein processing protein synthesis regulation review signal transduction ubiquitination Kang, J Pervaiz, S Crosstalk between Bcl-2 family and Ras family small GTPases: potential cell fate regulation? |
description |
10.3389/fonc.2012.00206 |
author2 |
PHYSIOLOGY |
author_facet |
PHYSIOLOGY Kang, J Pervaiz, S |
format |
Review |
author |
Kang, J Pervaiz, S |
author_sort |
Kang, J |
title |
Crosstalk between Bcl-2 family and Ras family small GTPases: potential cell fate regulation? |
title_short |
Crosstalk between Bcl-2 family and Ras family small GTPases: potential cell fate regulation? |
title_full |
Crosstalk between Bcl-2 family and Ras family small GTPases: potential cell fate regulation? |
title_fullStr |
Crosstalk between Bcl-2 family and Ras family small GTPases: potential cell fate regulation? |
title_full_unstemmed |
Crosstalk between Bcl-2 family and Ras family small GTPases: potential cell fate regulation? |
title_sort |
crosstalk between bcl-2 family and ras family small gtpases: potential cell fate regulation? |
publishDate |
2020 |
url |
https://scholarbank.nus.edu.sg/handle/10635/176179 |
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1795374411202691072 |