Rapamycin-induced inhibition of HTLV-I LTR activity is rescued by c-Myb

10.1186/1742-4690-4-24

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Main Authors: Rose, N.J, Lever, A.M.L
Other Authors: MEDICINE
Format: Article
Published: BMC 2020
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Online Access:https://scholarbank.nus.edu.sg/handle/10635/178354
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spelling sg-nus-scholar.10635-1783542024-04-24T05:53:24Z Rapamycin-induced inhibition of HTLV-I LTR activity is rescued by c-Myb Rose, N.J Lever, A.M.L MEDICINE CCAAT enhancer binding protein immunoglobulin enhancer binding protein mammalian target of rapamycin messenger RNA protein c Myb rapamycin tacrolimus Tax protein transcription factor transcription factor Ets transcription factor Ets 1 transcription factor Ets 2 chloramphenicol acetyltransferase immunoglobulin enhancer binding protein protein c Myb rapamycin 5' untranslated region antibiotic sensitivity antiviral activity article combination chemotherapy complex formation concentration response controlled study deletion mutant down regulation gene expression regulation gene overexpression Human T cell leukemia virus 1 in vitro study long terminal repeat monotherapy nonhuman promoter region signal transduction transcription initiation wild type biosynthesis drug antagonism drug effect gene fusion genetic transcription genetics human long terminal repeat physiology reporter gene Retrovirus Human T-lymphotropic virus 1 Mammalia Artificial Gene Fusion Chloramphenicol O-Acetyltransferase Deltaretrovirus Genes, Reporter Human T-lymphotropic virus 1 Humans NF-kappa B Proto-Oncogene Proteins c-myb Sirolimus Terminal Repeat Sequences Transcription, Genetic 10.1186/1742-4690-4-24 Retrovirology 4 24 2020-10-20T09:30:39Z 2020-10-20T09:30:39Z 2007 Article Rose, N.J, Lever, A.M.L (2007). Rapamycin-induced inhibition of HTLV-I LTR activity is rescued by c-Myb. Retrovirology 4 : 24. ScholarBank@NUS Repository. https://doi.org/10.1186/1742-4690-4-24 1742-4690 https://scholarbank.nus.edu.sg/handle/10635/178354 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ BMC Unpaywall 20201031
institution National University of Singapore
building NUS Library
continent Asia
country Singapore
Singapore
content_provider NUS Library
collection ScholarBank@NUS
topic CCAAT enhancer binding protein
immunoglobulin enhancer binding protein
mammalian target of rapamycin
messenger RNA
protein c Myb
rapamycin
tacrolimus
Tax protein
transcription factor
transcription factor Ets
transcription factor Ets 1
transcription factor Ets 2
chloramphenicol acetyltransferase
immunoglobulin enhancer binding protein
protein c Myb
rapamycin
5' untranslated region
antibiotic sensitivity
antiviral activity
article
combination chemotherapy
complex formation
concentration response
controlled study
deletion mutant
down regulation
gene expression regulation
gene overexpression
Human T cell leukemia virus 1
in vitro study
long terminal repeat
monotherapy
nonhuman
promoter region
signal transduction
transcription initiation
wild type
biosynthesis
drug antagonism
drug effect
gene fusion
genetic transcription
genetics
human
long terminal repeat
physiology
reporter gene
Retrovirus
Human T-lymphotropic virus 1
Mammalia
Artificial Gene Fusion
Chloramphenicol O-Acetyltransferase
Deltaretrovirus
Genes, Reporter
Human T-lymphotropic virus 1
Humans
NF-kappa B
Proto-Oncogene Proteins c-myb
Sirolimus
Terminal Repeat Sequences
Transcription, Genetic
spellingShingle CCAAT enhancer binding protein
immunoglobulin enhancer binding protein
mammalian target of rapamycin
messenger RNA
protein c Myb
rapamycin
tacrolimus
Tax protein
transcription factor
transcription factor Ets
transcription factor Ets 1
transcription factor Ets 2
chloramphenicol acetyltransferase
immunoglobulin enhancer binding protein
protein c Myb
rapamycin
5' untranslated region
antibiotic sensitivity
antiviral activity
article
combination chemotherapy
complex formation
concentration response
controlled study
deletion mutant
down regulation
gene expression regulation
gene overexpression
Human T cell leukemia virus 1
in vitro study
long terminal repeat
monotherapy
nonhuman
promoter region
signal transduction
transcription initiation
wild type
biosynthesis
drug antagonism
drug effect
gene fusion
genetic transcription
genetics
human
long terminal repeat
physiology
reporter gene
Retrovirus
Human T-lymphotropic virus 1
Mammalia
Artificial Gene Fusion
Chloramphenicol O-Acetyltransferase
Deltaretrovirus
Genes, Reporter
Human T-lymphotropic virus 1
Humans
NF-kappa B
Proto-Oncogene Proteins c-myb
Sirolimus
Terminal Repeat Sequences
Transcription, Genetic
Rose, N.J
Lever, A.M.L
Rapamycin-induced inhibition of HTLV-I LTR activity is rescued by c-Myb
description 10.1186/1742-4690-4-24
author2 MEDICINE
author_facet MEDICINE
Rose, N.J
Lever, A.M.L
format Article
author Rose, N.J
Lever, A.M.L
author_sort Rose, N.J
title Rapamycin-induced inhibition of HTLV-I LTR activity is rescued by c-Myb
title_short Rapamycin-induced inhibition of HTLV-I LTR activity is rescued by c-Myb
title_full Rapamycin-induced inhibition of HTLV-I LTR activity is rescued by c-Myb
title_fullStr Rapamycin-induced inhibition of HTLV-I LTR activity is rescued by c-Myb
title_full_unstemmed Rapamycin-induced inhibition of HTLV-I LTR activity is rescued by c-Myb
title_sort rapamycin-induced inhibition of htlv-i ltr activity is rescued by c-myb
publisher BMC
publishDate 2020
url https://scholarbank.nus.edu.sg/handle/10635/178354
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