Candida albicans gains azole resistance by altering sphingolipid composition

10.1038/s41467-018-06944-1

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Main Authors:	Gao, J, Wang, H, Li, Z, Wong, A.H.-H, Wang, Y.-Z, Guo, Y, Lin, X, Zeng, G, Wang, Y, Wang, J
Other Authors:	BIOCHEMISTRY
Format:	Article
Published:	Nature Publishing Group 2020
Subjects:	double stranded DNA fluconazole fungal DNA genomic DNA piggybac transposase sphingolipid transposase unclassified drug very long chain fatty acid pyrrole derivative sterol chemical composition drug resistance fatty acid fungus genetic analysis genome lipid mass spectrometry mutagenicity mutation pesticide resistance antifungal resistance Article Candida albicans controlled study DNA content fatty acid synthesis fungal genome fungal strain genetic screening lipid composition lipogenesis mutagenesis nonhuman nuclear localization signal transposon upregulation drug effect fungal gene genetics haploidy metabolism nucleotide sequence physiology Azoles Base Sequence DNA Transposable Elements Drug Resistance, Fungal Genes, Fungal Genetic Testing Haploidy Mutagenesis Mutation Sphingolipids Sterols
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/178388
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Institution:	National University of Singapore

id	sg-nus-scholar.10635-178388
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spelling	sg-nus-scholar.10635-1783882024-04-22T03:16:31Z Candida albicans gains azole resistance by altering sphingolipid composition Gao, J Wang, H Li, Z Wong, A.H.-H Wang, Y.-Z Guo, Y Lin, X Zeng, G Wang, Y Wang, J BIOCHEMISTRY double stranded DNA fluconazole fungal DNA genomic DNA piggybac transposase sphingolipid transposase unclassified drug very long chain fatty acid pyrrole derivative sphingolipid sterol chemical composition drug resistance fatty acid fungus genetic analysis genome lipid mass spectrometry mutagenicity mutation pesticide resistance antifungal resistance Article Candida albicans controlled study DNA content fatty acid synthesis fungal genome fungal strain genetic screening lipid composition lipogenesis mass spectrometry mutagenesis nonhuman nuclear localization signal transposon upregulation antifungal resistance Candida albicans drug effect fungal gene genetics haploidy metabolism mutation nucleotide sequence physiology Candida albicans Azoles Base Sequence Candida albicans DNA Transposable Elements Drug Resistance, Fungal Genes, Fungal Genetic Testing Haploidy Mutagenesis Mutation Sphingolipids Sterols 10.1038/s41467-018-06944-1 Nature Communications 9 1 4495 2020-10-20T09:40:36Z 2020-10-20T09:40:36Z 2018 Article Gao, J, Wang, H, Li, Z, Wong, A.H.-H, Wang, Y.-Z, Guo, Y, Lin, X, Zeng, G, Wang, Y, Wang, J (2018). Candida albicans gains azole resistance by altering sphingolipid composition. Nature Communications 9 (1) : 4495. ScholarBank@NUS Repository. https://doi.org/10.1038/s41467-018-06944-1 2041-1723 https://scholarbank.nus.edu.sg/handle/10635/178388 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ Nature Publishing Group Unpaywall 20201031
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	double stranded DNA fluconazole fungal DNA genomic DNA piggybac transposase sphingolipid transposase unclassified drug very long chain fatty acid pyrrole derivative sphingolipid sterol chemical composition drug resistance fatty acid fungus genetic analysis genome lipid mass spectrometry mutagenicity mutation pesticide resistance antifungal resistance Article Candida albicans controlled study DNA content fatty acid synthesis fungal genome fungal strain genetic screening lipid composition lipogenesis mass spectrometry mutagenesis nonhuman nuclear localization signal transposon upregulation antifungal resistance Candida albicans drug effect fungal gene genetics haploidy metabolism mutation nucleotide sequence physiology Candida albicans Azoles Base Sequence Candida albicans DNA Transposable Elements Drug Resistance, Fungal Genes, Fungal Genetic Testing Haploidy Mutagenesis Mutation Sphingolipids Sterols
spellingShingle	double stranded DNA fluconazole fungal DNA genomic DNA piggybac transposase sphingolipid transposase unclassified drug very long chain fatty acid pyrrole derivative sphingolipid sterol chemical composition drug resistance fatty acid fungus genetic analysis genome lipid mass spectrometry mutagenicity mutation pesticide resistance antifungal resistance Article Candida albicans controlled study DNA content fatty acid synthesis fungal genome fungal strain genetic screening lipid composition lipogenesis mass spectrometry mutagenesis nonhuman nuclear localization signal transposon upregulation antifungal resistance Candida albicans drug effect fungal gene genetics haploidy metabolism mutation nucleotide sequence physiology Candida albicans Azoles Base Sequence Candida albicans DNA Transposable Elements Drug Resistance, Fungal Genes, Fungal Genetic Testing Haploidy Mutagenesis Mutation Sphingolipids Sterols Gao, J Wang, H Li, Z Wong, A.H.-H Wang, Y.-Z Guo, Y Lin, X Zeng, G Wang, Y Wang, J Candida albicans gains azole resistance by altering sphingolipid composition
description	10.1038/s41467-018-06944-1
author2	BIOCHEMISTRY
author_facet	BIOCHEMISTRY Gao, J Wang, H Li, Z Wong, A.H.-H Wang, Y.-Z Guo, Y Lin, X Zeng, G Wang, Y Wang, J
format	Article
author	Gao, J Wang, H Li, Z Wong, A.H.-H Wang, Y.-Z Guo, Y Lin, X Zeng, G Wang, Y Wang, J
author_sort	Gao, J
title	Candida albicans gains azole resistance by altering sphingolipid composition
title_short	Candida albicans gains azole resistance by altering sphingolipid composition
title_full	Candida albicans gains azole resistance by altering sphingolipid composition
title_fullStr	Candida albicans gains azole resistance by altering sphingolipid composition
title_full_unstemmed	Candida albicans gains azole resistance by altering sphingolipid composition
title_sort	candida albicans gains azole resistance by altering sphingolipid composition
publisher	Nature Publishing Group
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/178388
_version_	1800914495288115200

Candida albicans gains azole resistance by altering sphingolipid composition

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