Integrative proteomics in prostate cancer uncovers robustness against genomic and transcriptomic aberrations during disease progression

10.1038/s41467-018-03573-6

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Main Authors: Latonen, L, Afyounian, E, Jylhä, A, Nättinen, J, Aapola, U, Annala, M, Kivinummi, K.K, Tammela, T.T.L, Beuerman, R.W, Uusitalo, H, Nykter, M, Visakorpi, T
Other Authors: DUKE-NUS MEDICAL SCHOOL
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Published: Nature Publishing Group 2020
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Online Access:https://scholarbank.nus.edu.sg/handle/10635/178420
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spelling sg-nus-scholar.10635-1784202023-11-01T09:02:54Z Integrative proteomics in prostate cancer uncovers robustness against genomic and transcriptomic aberrations during disease progression Latonen, L Afyounian, E Jylhä, A Nättinen, J Aapola, U Annala, M Kivinummi, K.K Tammela, T.T.L Beuerman, R.W Uusitalo, H Nykter, M Visakorpi, T DUKE-NUS MEDICAL SCHOOL messenger RNA microRNA proteome messenger RNA microRNA transcriptome tumor protein cancer castration citric acid gene expression genetic analysis methylation protein proteomics adult aged Article cancer growth castration resistant prostate cancer citric acid cycle controlled study DNA methylation gene dosage gene expression level genomics human human tissue male mass spectrometry prostate cancer prostate hypertrophy proteogenomics proteomics transcriptomics disease exacerbation gene expression regulation genetics genome-wide association study genomics metabolism middle aged pathology prostate Aged Citric Acid Cycle Disease Progression DNA Methylation Gene Dosage Gene Expression Regulation, Neoplastic Genome-Wide Association Study Genomics Humans Male MicroRNAs Middle Aged Neoplasm Proteins Prostate Prostatic Hyperplasia Prostatic Neoplasms, Castration-Resistant RNA, Messenger Transcriptome 10.1038/s41467-018-03573-6 Nature Communications 9 1 1176 2020-10-20T09:52:44Z 2020-10-20T09:52:44Z 2018 Article Latonen, L, Afyounian, E, Jylhä, A, Nättinen, J, Aapola, U, Annala, M, Kivinummi, K.K, Tammela, T.T.L, Beuerman, R.W, Uusitalo, H, Nykter, M, Visakorpi, T (2018). Integrative proteomics in prostate cancer uncovers robustness against genomic and transcriptomic aberrations during disease progression. Nature Communications 9 (1) : 1176. ScholarBank@NUS Repository. https://doi.org/10.1038/s41467-018-03573-6 2041-1723 https://scholarbank.nus.edu.sg/handle/10635/178420 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ Nature Publishing Group Unpaywall 20201031
institution National University of Singapore
building NUS Library
continent Asia
country Singapore
Singapore
content_provider NUS Library
collection ScholarBank@NUS
topic messenger RNA
microRNA
proteome
messenger RNA
microRNA
transcriptome
tumor protein
cancer
castration
citric acid
gene expression
genetic analysis
methylation
protein
proteomics
adult
aged
Article
cancer growth
castration resistant prostate cancer
citric acid cycle
controlled study
DNA methylation
gene dosage
gene expression level
genomics
human
human tissue
male
mass spectrometry
prostate cancer
prostate hypertrophy
proteogenomics
proteomics
transcriptomics
disease exacerbation
gene expression regulation
genetics
genome-wide association study
genomics
metabolism
middle aged
pathology
prostate
Aged
Citric Acid Cycle
Disease Progression
DNA Methylation
Gene Dosage
Gene Expression Regulation, Neoplastic
Genome-Wide Association Study
Genomics
Humans
Male
MicroRNAs
Middle Aged
Neoplasm Proteins
Prostate
Prostatic Hyperplasia
Prostatic Neoplasms, Castration-Resistant
RNA, Messenger
Transcriptome
spellingShingle messenger RNA
microRNA
proteome
messenger RNA
microRNA
transcriptome
tumor protein
cancer
castration
citric acid
gene expression
genetic analysis
methylation
protein
proteomics
adult
aged
Article
cancer growth
castration resistant prostate cancer
citric acid cycle
controlled study
DNA methylation
gene dosage
gene expression level
genomics
human
human tissue
male
mass spectrometry
prostate cancer
prostate hypertrophy
proteogenomics
proteomics
transcriptomics
disease exacerbation
gene expression regulation
genetics
genome-wide association study
genomics
metabolism
middle aged
pathology
prostate
Aged
Citric Acid Cycle
Disease Progression
DNA Methylation
Gene Dosage
Gene Expression Regulation, Neoplastic
Genome-Wide Association Study
Genomics
Humans
Male
MicroRNAs
Middle Aged
Neoplasm Proteins
Prostate
Prostatic Hyperplasia
Prostatic Neoplasms, Castration-Resistant
RNA, Messenger
Transcriptome
Latonen, L
Afyounian, E
Jylhä, A
Nättinen, J
Aapola, U
Annala, M
Kivinummi, K.K
Tammela, T.T.L
Beuerman, R.W
Uusitalo, H
Nykter, M
Visakorpi, T
Integrative proteomics in prostate cancer uncovers robustness against genomic and transcriptomic aberrations during disease progression
description 10.1038/s41467-018-03573-6
author2 DUKE-NUS MEDICAL SCHOOL
author_facet DUKE-NUS MEDICAL SCHOOL
Latonen, L
Afyounian, E
Jylhä, A
Nättinen, J
Aapola, U
Annala, M
Kivinummi, K.K
Tammela, T.T.L
Beuerman, R.W
Uusitalo, H
Nykter, M
Visakorpi, T
format Article
author Latonen, L
Afyounian, E
Jylhä, A
Nättinen, J
Aapola, U
Annala, M
Kivinummi, K.K
Tammela, T.T.L
Beuerman, R.W
Uusitalo, H
Nykter, M
Visakorpi, T
author_sort Latonen, L
title Integrative proteomics in prostate cancer uncovers robustness against genomic and transcriptomic aberrations during disease progression
title_short Integrative proteomics in prostate cancer uncovers robustness against genomic and transcriptomic aberrations during disease progression
title_full Integrative proteomics in prostate cancer uncovers robustness against genomic and transcriptomic aberrations during disease progression
title_fullStr Integrative proteomics in prostate cancer uncovers robustness against genomic and transcriptomic aberrations during disease progression
title_full_unstemmed Integrative proteomics in prostate cancer uncovers robustness against genomic and transcriptomic aberrations during disease progression
title_sort integrative proteomics in prostate cancer uncovers robustness against genomic and transcriptomic aberrations during disease progression
publisher Nature Publishing Group
publishDate 2020
url https://scholarbank.nus.edu.sg/handle/10635/178420
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