The pro-inflammatory phenotype of the human non-classical monocyte subset is attributed to senescence article

10.1038/s41419-018-0327-1

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Main Authors:	Ong, S.-M, Hadadi, E, Dang, T.-M, Yeap, W.-H, Tan, C.T.-Y, Ng, T.-P, Larbi, A, Wong, S.-C
Other Authors:	PSYCHOLOGICAL MEDICINE
Format:	Article
Published:	Nature Publishing Group 2020
Subjects:	CD14 antigen CD16 antigen immunoglobulin enhancer binding protein interleukin 1alpha interleukin 1beta interleukin 6 interleukin 8 Ki 67 antigen macrophage inflammatory protein 1alpha macrophage inflammatory protein 1beta matrix metalloproteinase microRNA 146a mitogen activated protein kinase RANTES reactive oxygen metabolite synaptotagmin I toll like receptor tumor necrosis factor adult antigen expression Article atherosclerosis blood level cell aging cell proliferation cytokine production gene expression human human cell in vitro study inflammation mitochondrial membrane potential monocyte osteoarthritis peripheral blood mononuclear cell phenotype priority journal stimulus response telomere length
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/178538
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Institution:	National University of Singapore

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spelling	sg-nus-scholar.10635-1785382024-04-03T08:02:13Z The pro-inflammatory phenotype of the human non-classical monocyte subset is attributed to senescence article Ong, S.-M Hadadi, E Dang, T.-M Yeap, W.-H Tan, C.T.-Y Ng, T.-P Larbi, A Wong, S.-C PSYCHOLOGICAL MEDICINE MICROBIOLOGY AND IMMUNOLOGY CD14 antigen CD16 antigen immunoglobulin enhancer binding protein interleukin 1alpha interleukin 1beta interleukin 6 interleukin 8 Ki 67 antigen macrophage inflammatory protein 1alpha macrophage inflammatory protein 1beta matrix metalloproteinase microRNA 146a mitogen activated protein kinase RANTES reactive oxygen metabolite synaptotagmin I toll like receptor tumor necrosis factor adult antigen expression Article atherosclerosis blood level cell aging cell proliferation cytokine production gene expression human human cell in vitro study inflammation mitochondrial membrane potential monocyte osteoarthritis peripheral blood mononuclear cell phenotype priority journal stimulus response telomere length 10.1038/s41419-018-0327-1 Cell Death and Disease 9 3 266 2020-10-20T10:13:07Z 2020-10-20T10:13:07Z 2018 Article Ong, S.-M, Hadadi, E, Dang, T.-M, Yeap, W.-H, Tan, C.T.-Y, Ng, T.-P, Larbi, A, Wong, S.-C (2018). The pro-inflammatory phenotype of the human non-classical monocyte subset is attributed to senescence article. Cell Death and Disease 9 (3) : 266. ScholarBank@NUS Repository. https://doi.org/10.1038/s41419-018-0327-1 2041-4889 https://scholarbank.nus.edu.sg/handle/10635/178538 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ Nature Publishing Group Unpaywall 20201031
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	CD14 antigen CD16 antigen immunoglobulin enhancer binding protein interleukin 1alpha interleukin 1beta interleukin 6 interleukin 8 Ki 67 antigen macrophage inflammatory protein 1alpha macrophage inflammatory protein 1beta matrix metalloproteinase microRNA 146a mitogen activated protein kinase RANTES reactive oxygen metabolite synaptotagmin I toll like receptor tumor necrosis factor adult antigen expression Article atherosclerosis blood level cell aging cell proliferation cytokine production gene expression human human cell in vitro study inflammation mitochondrial membrane potential monocyte osteoarthritis peripheral blood mononuclear cell phenotype priority journal stimulus response telomere length
spellingShingle	CD14 antigen CD16 antigen immunoglobulin enhancer binding protein interleukin 1alpha interleukin 1beta interleukin 6 interleukin 8 Ki 67 antigen macrophage inflammatory protein 1alpha macrophage inflammatory protein 1beta matrix metalloproteinase microRNA 146a mitogen activated protein kinase RANTES reactive oxygen metabolite synaptotagmin I toll like receptor tumor necrosis factor adult antigen expression Article atherosclerosis blood level cell aging cell proliferation cytokine production gene expression human human cell in vitro study inflammation mitochondrial membrane potential monocyte osteoarthritis peripheral blood mononuclear cell phenotype priority journal stimulus response telomere length Ong, S.-M Hadadi, E Dang, T.-M Yeap, W.-H Tan, C.T.-Y Ng, T.-P Larbi, A Wong, S.-C The pro-inflammatory phenotype of the human non-classical monocyte subset is attributed to senescence article
description	10.1038/s41419-018-0327-1
author2	PSYCHOLOGICAL MEDICINE
author_facet	PSYCHOLOGICAL MEDICINE Ong, S.-M Hadadi, E Dang, T.-M Yeap, W.-H Tan, C.T.-Y Ng, T.-P Larbi, A Wong, S.-C
format	Article
author	Ong, S.-M Hadadi, E Dang, T.-M Yeap, W.-H Tan, C.T.-Y Ng, T.-P Larbi, A Wong, S.-C
author_sort	Ong, S.-M
title	The pro-inflammatory phenotype of the human non-classical monocyte subset is attributed to senescence article
title_short	The pro-inflammatory phenotype of the human non-classical monocyte subset is attributed to senescence article
title_full	The pro-inflammatory phenotype of the human non-classical monocyte subset is attributed to senescence article
title_fullStr	The pro-inflammatory phenotype of the human non-classical monocyte subset is attributed to senescence article
title_full_unstemmed	The pro-inflammatory phenotype of the human non-classical monocyte subset is attributed to senescence article
title_sort	pro-inflammatory phenotype of the human non-classical monocyte subset is attributed to senescence article
publisher	Nature Publishing Group
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/178538
_version_	1795374465249443840

The pro-inflammatory phenotype of the human non-classical monocyte subset is attributed to senescence article

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