Mitochondrial mutations drive prostate cancer aggression

• المؤلفون الرئيسيون: Hopkins, J.F, Sabelnykova, V.Y, Weischenfeldt, J, Simon, R, Aguiar, J.A, Alkallas, R, Heisler, L.E, Zhang, J, Watson, J.D, Chua, M.L.K, Fraser, M, Favero, F, Lawerenz, C, Plass, C, Sauter, G, McPherson, J.D, Van Der Kwast, T, Korbel, J, Schlomm, T, Bristow, R.G, Boutros, P.C
• مؤلفون آخرون: DUKE-NUS MEDICAL SCHOOL
• التنسيق: مقال
• منشور في: Nature Publishing Group 2020
• الموضوعات: mitochondrial DNA; cancer; disease incidence; genome; mitochondrion; tumor; adult; age; androgen deprivation therapy; Article; cancer growth; cancer localization; cancer radiotherapy; cancer survival; cohort analysis; controlled study; gene control; gene dosage; gene interaction; gene locus; gene sequence; Gleason score; human; human tissue; major clinical study; male; mitochondrial gene; mitochondrial genome; oncogene myc; prostate cancer; prostatectomy; single nucleotide polymorphism; somatic mutation; adenocarcinoma; aged; genetic association study; genetics; middle aged; pathology; point mutation; prostate tumor; survival analysis; tumor invasion; very elderly; Adenocarcinoma; Adult; Age Factors; Aged; Aged, 80 and over; DNA, Mitochondrial; Genes, myc; Genetic Association Studies; Genome, Mitochondrial; Humans; Male; Middle Aged; Neoplasm Invasiveness; Point Mutation; Prostatic Neoplasms; Survival Analysis
• الوصول للمادة أونلاين: https://scholarbank.nus.edu.sg/handle/10635/178575

10.1038/s41467-017-00377-y