Tau exacerbates excitotoxic brain damage in an animal model of stroke

10.1038/s41467-017-00618-0

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Main Authors:	Bi, M, Gladbach, A, Van Eersel, J, Ittner, A, Przybyla, M, Van Hummel, A, Chua, S.W, Hoven, J.V.D, Lee, W.S, Müller, J, Parmar, J, Jonquieres, G.V, Stefen, H, Guccione, E, Fath, T, Housley, G.D, Klugmann, M, Ke, Y.D, Ittner, L.M
Other Authors:	BIOCHEMISTRY
Format:	Article
Published:	Nature Publishing Group 2020
Subjects:	glutamic acid guanosine triphosphatase activating protein mitogen activated protein kinase Ras protein SynGAP1 protein tau protein unclassified drug Syngap1 protein, mouse animal brain compartmentalization experimental study gene expression inhibition protein stroke toxicity animal experiment animal model Article brain damage cerebrovascular accident controlled study disease exacerbation enzyme activation excitotoxicity gene overexpression middle cerebral artery occlusion mouse nerve cell nonhuman postsynaptic density reperfusion signal transduction wild type brain injury C57BL mouse cell culture cerebral artery disease complication deficiency disease model gene expression profiling genetics human knockout mouse male metabolism procedures synaptosome Animalia Mus Animals Brain Injuries Cells, Cultured Disease Models, Animal Gene Expression Profiling Humans Infarction, Middle Cerebral Artery Male Mice, Inbred C57BL Mice, Knockout Neurons ras GTPase-Activating Proteins Signal Transduction Stroke Synaptosomes tau Proteins
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/178581
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Institution:	National University of Singapore

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spelling	sg-nus-scholar.10635-1785812024-04-22T03:33:50Z Tau exacerbates excitotoxic brain damage in an animal model of stroke Bi, M Gladbach, A Van Eersel, J Ittner, A Przybyla, M Van Hummel, A Chua, S.W Hoven, J.V.D Lee, W.S Müller, J Parmar, J Jonquieres, G.V Stefen, H Guccione, E Fath, T Housley, G.D Klugmann, M Ke, Y.D Ittner, L.M BIOCHEMISTRY glutamic acid guanosine triphosphatase activating protein mitogen activated protein kinase Ras protein SynGAP1 protein tau protein unclassified drug guanosine triphosphatase activating protein Syngap1 protein, mouse tau protein animal brain compartmentalization experimental study gene expression inhibition protein stroke toxicity animal experiment animal model Article brain damage cerebrovascular accident controlled study disease exacerbation enzyme activation excitotoxicity gene overexpression middle cerebral artery occlusion mouse nerve cell nonhuman postsynaptic density reperfusion signal transduction wild type animal brain injury C57BL mouse cell culture cerebral artery disease cerebrovascular accident complication deficiency disease model gene expression profiling genetics human knockout mouse male metabolism procedures synaptosome Animalia Mus Animals Brain Injuries Cells, Cultured Disease Models, Animal Gene Expression Profiling Humans Infarction, Middle Cerebral Artery Male Mice, Inbred C57BL Mice, Knockout Neurons ras GTPase-Activating Proteins Signal Transduction Stroke Synaptosomes tau Proteins 10.1038/s41467-017-00618-0 Nature Communications 8 1 473 2020-10-20T10:24:49Z 2020-10-20T10:24:49Z 2017 Article Bi, M, Gladbach, A, Van Eersel, J, Ittner, A, Przybyla, M, Van Hummel, A, Chua, S.W, Hoven, J.V.D, Lee, W.S, Müller, J, Parmar, J, Jonquieres, G.V, Stefen, H, Guccione, E, Fath, T, Housley, G.D, Klugmann, M, Ke, Y.D, Ittner, L.M (2017). Tau exacerbates excitotoxic brain damage in an animal model of stroke. Nature Communications 8 (1) : 473. ScholarBank@NUS Repository. https://doi.org/10.1038/s41467-017-00618-0 2041-1723 https://scholarbank.nus.edu.sg/handle/10635/178581 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ Nature Publishing Group Unpaywall 20201031
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	glutamic acid guanosine triphosphatase activating protein mitogen activated protein kinase Ras protein SynGAP1 protein tau protein unclassified drug guanosine triphosphatase activating protein Syngap1 protein, mouse tau protein animal brain compartmentalization experimental study gene expression inhibition protein stroke toxicity animal experiment animal model Article brain damage cerebrovascular accident controlled study disease exacerbation enzyme activation excitotoxicity gene overexpression middle cerebral artery occlusion mouse nerve cell nonhuman postsynaptic density reperfusion signal transduction wild type animal brain injury C57BL mouse cell culture cerebral artery disease cerebrovascular accident complication deficiency disease model gene expression profiling genetics human knockout mouse male metabolism procedures synaptosome Animalia Mus Animals Brain Injuries Cells, Cultured Disease Models, Animal Gene Expression Profiling Humans Infarction, Middle Cerebral Artery Male Mice, Inbred C57BL Mice, Knockout Neurons ras GTPase-Activating Proteins Signal Transduction Stroke Synaptosomes tau Proteins
spellingShingle	glutamic acid guanosine triphosphatase activating protein mitogen activated protein kinase Ras protein SynGAP1 protein tau protein unclassified drug guanosine triphosphatase activating protein Syngap1 protein, mouse tau protein animal brain compartmentalization experimental study gene expression inhibition protein stroke toxicity animal experiment animal model Article brain damage cerebrovascular accident controlled study disease exacerbation enzyme activation excitotoxicity gene overexpression middle cerebral artery occlusion mouse nerve cell nonhuman postsynaptic density reperfusion signal transduction wild type animal brain injury C57BL mouse cell culture cerebral artery disease cerebrovascular accident complication deficiency disease model gene expression profiling genetics human knockout mouse male metabolism procedures synaptosome Animalia Mus Animals Brain Injuries Cells, Cultured Disease Models, Animal Gene Expression Profiling Humans Infarction, Middle Cerebral Artery Male Mice, Inbred C57BL Mice, Knockout Neurons ras GTPase-Activating Proteins Signal Transduction Stroke Synaptosomes tau Proteins Bi, M Gladbach, A Van Eersel, J Ittner, A Przybyla, M Van Hummel, A Chua, S.W Hoven, J.V.D Lee, W.S Müller, J Parmar, J Jonquieres, G.V Stefen, H Guccione, E Fath, T Housley, G.D Klugmann, M Ke, Y.D Ittner, L.M Tau exacerbates excitotoxic brain damage in an animal model of stroke
description	10.1038/s41467-017-00618-0
author2	BIOCHEMISTRY
author_facet	BIOCHEMISTRY Bi, M Gladbach, A Van Eersel, J Ittner, A Przybyla, M Van Hummel, A Chua, S.W Hoven, J.V.D Lee, W.S Müller, J Parmar, J Jonquieres, G.V Stefen, H Guccione, E Fath, T Housley, G.D Klugmann, M Ke, Y.D Ittner, L.M
format	Article
author	Bi, M Gladbach, A Van Eersel, J Ittner, A Przybyla, M Van Hummel, A Chua, S.W Hoven, J.V.D Lee, W.S Müller, J Parmar, J Jonquieres, G.V Stefen, H Guccione, E Fath, T Housley, G.D Klugmann, M Ke, Y.D Ittner, L.M
author_sort	Bi, M
title	Tau exacerbates excitotoxic brain damage in an animal model of stroke
title_short	Tau exacerbates excitotoxic brain damage in an animal model of stroke
title_full	Tau exacerbates excitotoxic brain damage in an animal model of stroke
title_fullStr	Tau exacerbates excitotoxic brain damage in an animal model of stroke
title_full_unstemmed	Tau exacerbates excitotoxic brain damage in an animal model of stroke
title_sort	tau exacerbates excitotoxic brain damage in an animal model of stroke
publisher	Nature Publishing Group
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/178581
_version_	1800914510899314688

Tau exacerbates excitotoxic brain damage in an animal model of stroke

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