The activation of OR51E1 causes growth suppression of human prostate cancer cells

• المؤلفون الرئيسيون: Maßberg, D, Jovancevic, N, Offermann, A, Simon, A, Baniahmad, A, Perner, S, Pungsrinont, T, Luko, K, Philippou, S, Ubrig, B, Heiland, M, Weber, L, Altmüller, J, Becker, C, Gisselmann, G, Gelis, L, Hatt, H
• مؤلفون آخرون: DUKE-NUS MEDICAL SCHOOL
• التنسيق: مقال
• منشور في: 2020
• الموضوعات: amyl butyrate; androgen receptor; butyric acid derivative; G protein coupled receptor; messenger RNA; nonanoic acid; nonanol; prostate specific G protein coupled receptor; prostate specific G protein coupled receptor 2; protein kinase; RNA; transcription factor E2F1; unclassified drug; AR protein, human; OR51E1 protein, human; tumor protein; Article; cancer cell; cancer inhibition; cancer tissue; cell aging; cell proliferation; cellular distribution; controlled study; enzyme linked immunosorbent assay; enzyme phosphorylation; gene expression; genetic transfection; human; human cell; human tissue; immunocytochemistry; immunohistochemistry; LNCaP cell line; male; plasmid; prostate cancer; protein expression; protein localization; reverse transcription polymerase chain reaction; RNA isolation; RNA sequence; Western blotting; biosynthesis; castration resistant prostate cancer; disease exacerbation; genetics; metabolism; pathology; phosphorylation; physiology; prostate tumor; signal transduction; tumor cell line; Cell Line, Tumor; Cell Proliferation; Cellular Senescence; Disease Progression; Humans; Male; Neoplasm Proteins; Phosphorylation; Prostatic Neoplasms; Prostatic Neoplasms, Castration-Resistant; Receptors, Androgen; Receptors, G-Protein-Coupled; Signal Transduction; Transfection
• الوصول للمادة أونلاين: https://scholarbank.nus.edu.sg/handle/10635/179968

10.18632/oncotarget.10197