Loss of fas apoptosis inhibitory molecule leads to spontaneous obesity and hepatosteatosis

10.1038/cddis.2016.12

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Main Authors: Huo, J, Ma, Y, Liu, J.-J, Ho, Y.S, Liu, S, Soh, L.Y, Chen, S, Xu, S, Han, W, Hong, A, Lim, S.C, Lam, K.-P
Other Authors: PHYSIOLOGY
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Published: Nature Publishing Group 2020
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Online Access:https://scholarbank.nus.edu.sg/handle/10635/180385
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spelling sg-nus-scholar.10635-1803852024-04-04T01:09:04Z Loss of fas apoptosis inhibitory molecule leads to spontaneous obesity and hepatosteatosis Huo, J Ma, Y Liu, J.-J Ho, Y.S Liu, S Soh, L.Y Chen, S Xu, S Han, W Hong, A Lim, S.C Lam, K.-P PHYSIOLOGY DUKE-NUS MEDICAL SCHOOL MICROBIOLOGY AND IMMUNOLOGY apoptosis regulatory protein FAIM protein, human Faim protein, mouse insulin animal C57BL mouse deficiency energy metabolism female human knockout mouse lipogenesis male metabolism middle aged mouse nonalcoholic fatty liver obesity Animals Apoptosis Regulatory Proteins Energy Metabolism Female Humans Insulin Lipogenesis Male Mice Mice, Inbred C57BL Mice, Knockout Middle Aged Non-alcoholic Fatty Liver Disease Obesity 10.1038/cddis.2016.12 Cell Death and Disease 7 2 e2091 2020-10-26T08:44:49Z 2020-10-26T08:44:49Z 2016 Article Huo, J, Ma, Y, Liu, J.-J, Ho, Y.S, Liu, S, Soh, L.Y, Chen, S, Xu, S, Han, W, Hong, A, Lim, S.C, Lam, K.-P (2016). Loss of fas apoptosis inhibitory molecule leads to spontaneous obesity and hepatosteatosis. Cell Death and Disease 7 (2) : e2091. ScholarBank@NUS Repository. https://doi.org/10.1038/cddis.2016.12 2041-4889 https://scholarbank.nus.edu.sg/handle/10635/180385 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ Nature Publishing Group Unpaywall 20201031
institution National University of Singapore
building NUS Library
continent Asia
country Singapore
Singapore
content_provider NUS Library
collection ScholarBank@NUS
topic apoptosis regulatory protein
FAIM protein, human
Faim protein, mouse
insulin
animal
C57BL mouse
deficiency
energy metabolism
female
human
knockout mouse
lipogenesis
male
metabolism
middle aged
mouse
nonalcoholic fatty liver
obesity
Animals
Apoptosis Regulatory Proteins
Energy Metabolism
Female
Humans
Insulin
Lipogenesis
Male
Mice
Mice, Inbred C57BL
Mice, Knockout
Middle Aged
Non-alcoholic Fatty Liver Disease
Obesity
spellingShingle apoptosis regulatory protein
FAIM protein, human
Faim protein, mouse
insulin
animal
C57BL mouse
deficiency
energy metabolism
female
human
knockout mouse
lipogenesis
male
metabolism
middle aged
mouse
nonalcoholic fatty liver
obesity
Animals
Apoptosis Regulatory Proteins
Energy Metabolism
Female
Humans
Insulin
Lipogenesis
Male
Mice
Mice, Inbred C57BL
Mice, Knockout
Middle Aged
Non-alcoholic Fatty Liver Disease
Obesity
Huo, J
Ma, Y
Liu, J.-J
Ho, Y.S
Liu, S
Soh, L.Y
Chen, S
Xu, S
Han, W
Hong, A
Lim, S.C
Lam, K.-P
Loss of fas apoptosis inhibitory molecule leads to spontaneous obesity and hepatosteatosis
description 10.1038/cddis.2016.12
author2 PHYSIOLOGY
author_facet PHYSIOLOGY
Huo, J
Ma, Y
Liu, J.-J
Ho, Y.S
Liu, S
Soh, L.Y
Chen, S
Xu, S
Han, W
Hong, A
Lim, S.C
Lam, K.-P
format Article
author Huo, J
Ma, Y
Liu, J.-J
Ho, Y.S
Liu, S
Soh, L.Y
Chen, S
Xu, S
Han, W
Hong, A
Lim, S.C
Lam, K.-P
author_sort Huo, J
title Loss of fas apoptosis inhibitory molecule leads to spontaneous obesity and hepatosteatosis
title_short Loss of fas apoptosis inhibitory molecule leads to spontaneous obesity and hepatosteatosis
title_full Loss of fas apoptosis inhibitory molecule leads to spontaneous obesity and hepatosteatosis
title_fullStr Loss of fas apoptosis inhibitory molecule leads to spontaneous obesity and hepatosteatosis
title_full_unstemmed Loss of fas apoptosis inhibitory molecule leads to spontaneous obesity and hepatosteatosis
title_sort loss of fas apoptosis inhibitory molecule leads to spontaneous obesity and hepatosteatosis
publisher Nature Publishing Group
publishDate 2020
url https://scholarbank.nus.edu.sg/handle/10635/180385
_version_ 1795374521070387200