Activated macrophages promote hepatitis C virus entry in a tumor necrosis factor-dependent manner

• Main Authors: Fletcher, N.F, Sutaria, R, Jo, J, Barnes, A, Blahova, M, Meredith, L.W, Cosset, F.-L, Curbishley, S.M, Adams, D.H, Bertoletti, A, Mckeating, J.A
• Other Authors: DUKE-NUS MEDICAL SCHOOL
• Format: Article
• Published: John Wiley and Sons Ltd 2020
• Subjects: CD81 antigen; gamma interferon; interleukin 10; interleukin 17; interleukin 18; interleukin 1beta; interleukin 4; interleukin 6; interleukin 8; messenger RNA; occludin; tight junction protein; tumor necrosis factor alpha; article; cell differentiation; cell membrane permeability; cell strain HepG2; controlled study; diffusion coefficient; hepatitis C; Hepatitis C virus; hepatoma cell; human; human cell; Human immunodeficiency virus; innate immunity; Kupffer cell; Lassa virus; liver cell; macrophage activation; Measles virus; nonhuman; polarization; priority journal; vesicular stomatitis; Vesicular stomatitis virus; virus entry; Antigens, CD81; Carcinoma, Hepatocellular; Cell Line, Tumor; Cell Polarity; Hep G2 Cells; Hepacivirus; Hepatitis C; Humans; Immunity, Innate; Interleukin-1beta; Liver Neoplasms; Macrophage Activation; Macrophages; Occludin; Tight Junctions; Tumor Necrosis Factor-alpha; Virus Internalization
• Online Access: https://scholarbank.nus.edu.sg/handle/10635/180764
• Institution: National University of Singapore

10.1002/hep.26911