Comment on "Development of murine lupus involves the combined genetic contribution of the SLAM and Fc?R intervals within the Nba2 autoimmune susceptibility locus"

• Main Authors: Choubey, D, Panchanathan, R, Shen, H, Duan, X
• Other Authors: MICROBIOLOGY AND IMMUNOLOGY
• Format: Letter
• Published: 2020
• Subjects: alpha interferon; antinuclear antibody; beta 2 microglobulin; CD150 antigen; cell protein; Fc receptor; histone H3; interferon; protein p200; protein p53; transcription factor E2F1; unclassified drug; autacoid; cell surface receptor; Ifi202b protein, mouse; leukocyte antigen; signal peptide; antibody production; autoimmunity; B lymphocyte; cell fractionation; cellular distribution; gene locus; genetic susceptibility; immunoblotting; letter; lupus vulgaris; nonhuman; priority journal; protein analysis; protein localization; spleen cell; animal; C57BL mouse; genetic marker; genetic predisposition; genetics; immunology; lupus erythematosus nephritis; metabolism; mouse; New Zealand black mouse; note; physiology; Animals; Antibodies, Antinuclear; Antigens, CD; Genetic Markers; Genetic Predisposition to Disease; Inflammation Mediators; Intracellular Signaling Peptides and Proteins; Lupus Nephritis; Mice; Mice, Inbred C57BL; Mice, Inbred NZB; Receptors, Cell Surface; Receptors, IgG
• Online Access: https://scholarbank.nus.edu.sg/handle/10635/180993
• Institution: National University of Singapore

10.4049/jimmunol.1090015