Sirenomelia in Bmp7 and Tsg compound mutant mice: Requirement for Bmp signaling in the development of ventral posterior mesoderm

• المؤلفون الرئيسيون: Zakin, L, Reversade, B, Kuroda, H, Lyons, K.M, De Robertis, E.M
• مؤلفون آخرون: PAEDIATRICS
• التنسيق: مقال
• منشور في: 2020
• الموضوعات: bone morphogenetic protein; oligonucleotide; osteogenic protein 1; protein Tsg; unclassified drug; animal experiment; animal model; article; controlled study; embryo; extracellular space; gastrulation; gene mutation; mesoderm; mouse; nonhuman; phenotype; priority journal; protein binding; protein interaction; protein metabolism; signal transduction; sirenomelia; Xenopus; Animals; Blotting, Western; Body Patterning; Bone Morphogenetic Proteins; Ectromelia; Gene Expression Regulation, Developmental; Hindlimb; Histological Techniques; In Situ Hybridization; Mesoderm; Mice; Mutation; Oligonucleotides, Antisense; Proteins; Reverse Transcriptase Polymerase Chain Reaction; Rodent Diseases; Signal Transduction; Transforming Growth Factor beta; Animalia; Sirenidae; Vertebrata
• الوصول للمادة أونلاين: https://scholarbank.nus.edu.sg/handle/10635/181092

10.1242/dev.01822