The role of O-GlcNAcylation for protection against ischemia-reperfusion injury

10.3390/ijms20020404

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Main Authors:	Jensen, R.V, Andreadou, I, Hausenloy, D.J, Bøtker, H.E
Other Authors:	DUKE-NUS MEDICAL SCHOOL
Format:	Review
Published:	2020
Subjects:	calcium mitochondrial permeability transition pore n acetylglucosamine cardiotonic agent carrier protein reactive oxygen metabolite biosynthesis cell death cell survival cell viability diabetes mellitus endoplasmic reticulum stress fluorescence microscopy gene overexpression heart function heart protection heat shock response hemodynamics human inflammation ischemic heart disease ischemic preconditioning morbidity mortality nonhuman oxidative stress reperfusion injury Review signal transduction stress animal comorbidity drug effect metabolism myocardial ischemia reperfusion injury pathology protein processing Acetylglucosamine Animals Cardiotonic Agents Comorbidity Humans Metabolic Networks and Pathways Mitochondrial Membrane Transport Proteins Myocardial Reperfusion Injury Oxidative Stress Protein Processing, Post-Translational Reactive Oxygen Species Reperfusion Injury
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/183292
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Institution:	National University of Singapore

id	sg-nus-scholar.10635-183292
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spelling	sg-nus-scholar.10635-1832922023-09-20T08:40:17Z The role of O-GlcNAcylation for protection against ischemia-reperfusion injury Jensen, R.V Andreadou, I Hausenloy, D.J Bøtker, H.E DUKE-NUS MEDICAL SCHOOL calcium mitochondrial permeability transition pore n acetylglucosamine cardiotonic agent carrier protein n acetylglucosamine reactive oxygen metabolite biosynthesis cell death cell survival cell viability diabetes mellitus endoplasmic reticulum stress fluorescence microscopy gene overexpression heart function heart protection heat shock response hemodynamics human inflammation ischemic heart disease ischemic preconditioning morbidity mortality nonhuman oxidative stress reperfusion injury Review signal transduction stress animal comorbidity drug effect metabolism myocardial ischemia reperfusion injury oxidative stress pathology protein processing reperfusion injury Acetylglucosamine Animals Cardiotonic Agents Comorbidity Humans Metabolic Networks and Pathways Mitochondrial Membrane Transport Proteins Myocardial Reperfusion Injury Oxidative Stress Protein Processing, Post-Translational Reactive Oxygen Species Reperfusion Injury 10.3390/ijms20020404 International Journal of Molecular Sciences 20 2 404 2020-11-10T00:47:23Z 2020-11-10T00:47:23Z 2019 Review Jensen, R.V, Andreadou, I, Hausenloy, D.J, Bøtker, H.E (2019). The role of O-GlcNAcylation for protection against ischemia-reperfusion injury. International Journal of Molecular Sciences 20 (2) : 404. ScholarBank@NUS Repository. https://doi.org/10.3390/ijms20020404 16616596 https://scholarbank.nus.edu.sg/handle/10635/183292 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ Unpaywall 20201031
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	calcium mitochondrial permeability transition pore n acetylglucosamine cardiotonic agent carrier protein n acetylglucosamine reactive oxygen metabolite biosynthesis cell death cell survival cell viability diabetes mellitus endoplasmic reticulum stress fluorescence microscopy gene overexpression heart function heart protection heat shock response hemodynamics human inflammation ischemic heart disease ischemic preconditioning morbidity mortality nonhuman oxidative stress reperfusion injury Review signal transduction stress animal comorbidity drug effect metabolism myocardial ischemia reperfusion injury oxidative stress pathology protein processing reperfusion injury Acetylglucosamine Animals Cardiotonic Agents Comorbidity Humans Metabolic Networks and Pathways Mitochondrial Membrane Transport Proteins Myocardial Reperfusion Injury Oxidative Stress Protein Processing, Post-Translational Reactive Oxygen Species Reperfusion Injury
spellingShingle	calcium mitochondrial permeability transition pore n acetylglucosamine cardiotonic agent carrier protein n acetylglucosamine reactive oxygen metabolite biosynthesis cell death cell survival cell viability diabetes mellitus endoplasmic reticulum stress fluorescence microscopy gene overexpression heart function heart protection heat shock response hemodynamics human inflammation ischemic heart disease ischemic preconditioning morbidity mortality nonhuman oxidative stress reperfusion injury Review signal transduction stress animal comorbidity drug effect metabolism myocardial ischemia reperfusion injury oxidative stress pathology protein processing reperfusion injury Acetylglucosamine Animals Cardiotonic Agents Comorbidity Humans Metabolic Networks and Pathways Mitochondrial Membrane Transport Proteins Myocardial Reperfusion Injury Oxidative Stress Protein Processing, Post-Translational Reactive Oxygen Species Reperfusion Injury Jensen, R.V Andreadou, I Hausenloy, D.J Bøtker, H.E The role of O-GlcNAcylation for protection against ischemia-reperfusion injury
description	10.3390/ijms20020404
author2	DUKE-NUS MEDICAL SCHOOL
author_facet	DUKE-NUS MEDICAL SCHOOL Jensen, R.V Andreadou, I Hausenloy, D.J Bøtker, H.E
format	Review
author	Jensen, R.V Andreadou, I Hausenloy, D.J Bøtker, H.E
author_sort	Jensen, R.V
title	The role of O-GlcNAcylation for protection against ischemia-reperfusion injury
title_short	The role of O-GlcNAcylation for protection against ischemia-reperfusion injury
title_full	The role of O-GlcNAcylation for protection against ischemia-reperfusion injury
title_fullStr	The role of O-GlcNAcylation for protection against ischemia-reperfusion injury
title_full_unstemmed	The role of O-GlcNAcylation for protection against ischemia-reperfusion injury
title_sort	role of o-glcnacylation for protection against ischemia-reperfusion injury
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/183292
_version_	1779152994226405376

The role of O-GlcNAcylation for protection against ischemia-reperfusion injury

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