Transcription profiling of C/EBP targets identifies Per2 as a gene implicated in myeloid leukemia

10.1182/blood-2005-01-0358

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Main Authors:	Gery, S, Gombart, A.F., Yi, W.S., Koeffler, C., Hofmann, W.-K., Koeffler, H.P.
Other Authors:	MEDICINE
Format:	Article
Published:	American Society of Hematology 2020
Subjects:	CCAAT enhancer binding protein PER2 protein protein Rev protein unclassified drug acute granulocytic leukemia animal cell apoptosis article cell cycle cell differentiation cell growth cell strain 3T3 cell strain K 562 cell type controlled study DNA microarray gene gene expression gene identification gene period 2 gene targeting genetic transcription human human cell lymphoma cell line mouse myeloid leukemia nonhuman normal human nucleotide sequence priority journal transcription regulation unindexed sequence upregulation Animals Apoptosis CCAAT-Enhancer-Binding Proteins Cell Cycle Proteins Cell Proliferation Down-Regulation Gene Expression Profiling Gene Expression Regulation, Neoplastic Humans Leukemia, Myeloid Mice NIH 3T3 Cells Nuclear Proteins Oligonucleotide Array Sequence Analysis Transcription Factors Transcription, Genetic
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/183931
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Institution:	National University of Singapore

id	sg-nus-scholar.10635-183931
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spelling	sg-nus-scholar.10635-1839312024-05-08T09:50:57Z Transcription profiling of C/EBP targets identifies Per2 as a gene implicated in myeloid leukemia Gery, S Gombart, A.F. Yi, W.S. Koeffler, C. Hofmann, W.-K. Koeffler, H.P. MEDICINE CCAAT enhancer binding protein PER2 protein protein Rev protein unclassified drug acute granulocytic leukemia animal cell apoptosis article cell cycle cell differentiation cell growth cell strain 3T3 cell strain K 562 cell type controlled study DNA microarray gene gene expression gene identification gene period 2 gene targeting genetic transcription human human cell lymphoma cell line mouse myeloid leukemia nonhuman normal human nucleotide sequence priority journal transcription regulation unindexed sequence upregulation Animals Apoptosis CCAAT-Enhancer-Binding Proteins Cell Cycle Proteins Cell Proliferation Down-Regulation Gene Expression Profiling Gene Expression Regulation, Neoplastic Humans Leukemia, Myeloid Mice NIH 3T3 Cells Nuclear Proteins Oligonucleotide Array Sequence Analysis Transcription Factors Transcription, Genetic 10.1182/blood-2005-01-0358 Blood 106 8 2827-2836 2020-11-23T09:03:42Z 2020-11-23T09:03:42Z 2005 Article Gery, S, Gombart, A.F., Yi, W.S., Koeffler, C., Hofmann, W.-K., Koeffler, H.P. (2005). Transcription profiling of C/EBP targets identifies Per2 as a gene implicated in myeloid leukemia. Blood 106 (8) : 2827-2836. ScholarBank@NUS Repository. https://doi.org/10.1182/blood-2005-01-0358 0006-4971 https://scholarbank.nus.edu.sg/handle/10635/183931 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ American Society of Hematology Unpaywall 20201031
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	CCAAT enhancer binding protein PER2 protein protein Rev protein unclassified drug acute granulocytic leukemia animal cell apoptosis article cell cycle cell differentiation cell growth cell strain 3T3 cell strain K 562 cell type controlled study DNA microarray gene gene expression gene identification gene period 2 gene targeting genetic transcription human human cell lymphoma cell line mouse myeloid leukemia nonhuman normal human nucleotide sequence priority journal transcription regulation unindexed sequence upregulation Animals Apoptosis CCAAT-Enhancer-Binding Proteins Cell Cycle Proteins Cell Proliferation Down-Regulation Gene Expression Profiling Gene Expression Regulation, Neoplastic Humans Leukemia, Myeloid Mice NIH 3T3 Cells Nuclear Proteins Oligonucleotide Array Sequence Analysis Transcription Factors Transcription, Genetic
spellingShingle	CCAAT enhancer binding protein PER2 protein protein Rev protein unclassified drug acute granulocytic leukemia animal cell apoptosis article cell cycle cell differentiation cell growth cell strain 3T3 cell strain K 562 cell type controlled study DNA microarray gene gene expression gene identification gene period 2 gene targeting genetic transcription human human cell lymphoma cell line mouse myeloid leukemia nonhuman normal human nucleotide sequence priority journal transcription regulation unindexed sequence upregulation Animals Apoptosis CCAAT-Enhancer-Binding Proteins Cell Cycle Proteins Cell Proliferation Down-Regulation Gene Expression Profiling Gene Expression Regulation, Neoplastic Humans Leukemia, Myeloid Mice NIH 3T3 Cells Nuclear Proteins Oligonucleotide Array Sequence Analysis Transcription Factors Transcription, Genetic Gery, S Gombart, A.F. Yi, W.S. Koeffler, C. Hofmann, W.-K. Koeffler, H.P. Transcription profiling of C/EBP targets identifies Per2 as a gene implicated in myeloid leukemia
description	10.1182/blood-2005-01-0358
author2	MEDICINE
author_facet	MEDICINE Gery, S Gombart, A.F. Yi, W.S. Koeffler, C. Hofmann, W.-K. Koeffler, H.P.
format	Article
author	Gery, S Gombart, A.F. Yi, W.S. Koeffler, C. Hofmann, W.-K. Koeffler, H.P.
author_sort	Gery, S
title	Transcription profiling of C/EBP targets identifies Per2 as a gene implicated in myeloid leukemia
title_short	Transcription profiling of C/EBP targets identifies Per2 as a gene implicated in myeloid leukemia
title_full	Transcription profiling of C/EBP targets identifies Per2 as a gene implicated in myeloid leukemia
title_fullStr	Transcription profiling of C/EBP targets identifies Per2 as a gene implicated in myeloid leukemia
title_full_unstemmed	Transcription profiling of C/EBP targets identifies Per2 as a gene implicated in myeloid leukemia
title_sort	transcription profiling of c/ebp targets identifies per2 as a gene implicated in myeloid leukemia
publisher	American Society of Hematology
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/183931
_version_	1800914735408873472

Transcription profiling of C/EBP targets identifies Per2 as a gene implicated in myeloid leukemia

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