An Important Role for DNMT3A-Mediated DNA Methylation in Cardiomyocyte Metabolism and Contractility
10.1161/CIRCULATIONAHA.119.044444
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LIPPINCOTT WILLIAMS & WILKINS
2021
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sg-nus-scholar.10635-2059392024-04-18T01:59:49Z An Important Role for DNMT3A-Mediated DNA Methylation in Cardiomyocyte Metabolism and Contractility Madsen, Alexandra Hoeppner, Grit Krause, Julia Hirt, Marc N Laufer, Sandra D Schweizer, Michaela Tan, Wilson Lek Wen Mosqueira, Diogo Anene-Nzelu, Chukwuemeka George Lim, Ives Foo, Roger SY Eschenhagen, Thomas Stenzig, Justus MEDICINE Science & Technology Life Sciences & Biomedicine Cardiac & Cardiovascular Systems Peripheral Vascular Disease Cardiovascular System & Cardiology cardiac hypertrophy epigenetics tissue engineering INDUCIBLE FACTOR 1-ALPHA ENGINEERED HEART-TISSUE INDUCED CARDIAC-HYPERTROPHY GENE-EXPRESSION PPAR-GAMMA FAILURE PROTEIN PLURIPOTENT HIF-1-ALPHA INHIBITION 10.1161/CIRCULATIONAHA.119.044444 CIRCULATION 142 16 1562-1578 2021-11-11T07:58:05Z 2021-11-11T07:58:05Z 2020-10-20 2021-11-09T21:27:13Z Article Madsen, Alexandra, Hoeppner, Grit, Krause, Julia, Hirt, Marc N, Laufer, Sandra D, Schweizer, Michaela, Tan, Wilson Lek Wen, Mosqueira, Diogo, Anene-Nzelu, Chukwuemeka George, Lim, Ives, Foo, Roger SY, Eschenhagen, Thomas, Stenzig, Justus (2020-10-20). An Important Role for DNMT3A-Mediated DNA Methylation in Cardiomyocyte Metabolism and Contractility. CIRCULATION 142 (16) : 1562-1578. ScholarBank@NUS Repository. https://doi.org/10.1161/CIRCULATIONAHA.119.044444 00097322 15244539 https://scholarbank.nus.edu.sg/handle/10635/205939 en LIPPINCOTT WILLIAMS & WILKINS Elements |
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Science & Technology Life Sciences & Biomedicine Cardiac & Cardiovascular Systems Peripheral Vascular Disease Cardiovascular System & Cardiology cardiac hypertrophy epigenetics tissue engineering INDUCIBLE FACTOR 1-ALPHA ENGINEERED HEART-TISSUE INDUCED CARDIAC-HYPERTROPHY GENE-EXPRESSION PPAR-GAMMA FAILURE PROTEIN PLURIPOTENT HIF-1-ALPHA INHIBITION |
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Science & Technology Life Sciences & Biomedicine Cardiac & Cardiovascular Systems Peripheral Vascular Disease Cardiovascular System & Cardiology cardiac hypertrophy epigenetics tissue engineering INDUCIBLE FACTOR 1-ALPHA ENGINEERED HEART-TISSUE INDUCED CARDIAC-HYPERTROPHY GENE-EXPRESSION PPAR-GAMMA FAILURE PROTEIN PLURIPOTENT HIF-1-ALPHA INHIBITION Madsen, Alexandra Hoeppner, Grit Krause, Julia Hirt, Marc N Laufer, Sandra D Schweizer, Michaela Tan, Wilson Lek Wen Mosqueira, Diogo Anene-Nzelu, Chukwuemeka George Lim, Ives Foo, Roger SY Eschenhagen, Thomas Stenzig, Justus An Important Role for DNMT3A-Mediated DNA Methylation in Cardiomyocyte Metabolism and Contractility |
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10.1161/CIRCULATIONAHA.119.044444 |
author2 |
MEDICINE |
author_facet |
MEDICINE Madsen, Alexandra Hoeppner, Grit Krause, Julia Hirt, Marc N Laufer, Sandra D Schweizer, Michaela Tan, Wilson Lek Wen Mosqueira, Diogo Anene-Nzelu, Chukwuemeka George Lim, Ives Foo, Roger SY Eschenhagen, Thomas Stenzig, Justus |
format |
Article |
author |
Madsen, Alexandra Hoeppner, Grit Krause, Julia Hirt, Marc N Laufer, Sandra D Schweizer, Michaela Tan, Wilson Lek Wen Mosqueira, Diogo Anene-Nzelu, Chukwuemeka George Lim, Ives Foo, Roger SY Eschenhagen, Thomas Stenzig, Justus |
author_sort |
Madsen, Alexandra |
title |
An Important Role for DNMT3A-Mediated DNA Methylation in Cardiomyocyte Metabolism and Contractility |
title_short |
An Important Role for DNMT3A-Mediated DNA Methylation in Cardiomyocyte Metabolism and Contractility |
title_full |
An Important Role for DNMT3A-Mediated DNA Methylation in Cardiomyocyte Metabolism and Contractility |
title_fullStr |
An Important Role for DNMT3A-Mediated DNA Methylation in Cardiomyocyte Metabolism and Contractility |
title_full_unstemmed |
An Important Role for DNMT3A-Mediated DNA Methylation in Cardiomyocyte Metabolism and Contractility |
title_sort |
important role for dnmt3a-mediated dna methylation in cardiomyocyte metabolism and contractility |
publisher |
LIPPINCOTT WILLIAMS & WILKINS |
publishDate |
2021 |
url |
https://scholarbank.nus.edu.sg/handle/10635/205939 |
_version_ |
1800915044079239168 |