Diet-induced obesity, gut microbiota and bone, including alveolar bone loss

© 2017 Elsevier Ltd Obesity is a major risk factor for several pathologies, including jaw bone resorption. The underlying mechanisms involved in pathological conditions resulting from obesity include chronic systemic inflammation and the development of insulin resistance. Although numerous studies h...

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Main Authors: Sathima Eaimworawuthikul, Parameth Thiennimitr, Nipon Chattipakorn, Siriporn C. Chattipakorn
Format: Journal
Published: 2018
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Online Access:https://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=85012277820&origin=inward
http://cmuir.cmu.ac.th/jspui/handle/6653943832/46386
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Institution: Chiang Mai University
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spelling th-cmuir.6653943832-463862018-04-25T07:23:36Z Diet-induced obesity, gut microbiota and bone, including alveolar bone loss Sathima Eaimworawuthikul Parameth Thiennimitr Nipon Chattipakorn Siriporn C. Chattipakorn Dentistry Agricultural and Biological Sciences © 2017 Elsevier Ltd Obesity is a major risk factor for several pathologies, including jaw bone resorption. The underlying mechanisms involved in pathological conditions resulting from obesity include chronic systemic inflammation and the development of insulin resistance. Although numerous studies have indicated the importance of the role of gut microbiota in the pathogenesis of inflammation and insulin resistance in obesity, only a few studies have established a relationship between obesity, gut microbiota and status of the jaw bone. This review aims to summarize current findings relating to these issues, focusing on the role of obesity and gut microbiota on jaw bone health, including possible mechanisms which can explain this link. 2018-04-25T06:54:08Z 2018-04-25T06:54:08Z 2017-06-01 Journal 18791506 00039969 2-s2.0-85012277820 10.1016/j.archoralbio.2017.02.009 https://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=85012277820&origin=inward http://cmuir.cmu.ac.th/jspui/handle/6653943832/46386
institution Chiang Mai University
building Chiang Mai University Library
country Thailand
collection CMU Intellectual Repository
topic Dentistry
Agricultural and Biological Sciences
spellingShingle Dentistry
Agricultural and Biological Sciences
Sathima Eaimworawuthikul
Parameth Thiennimitr
Nipon Chattipakorn
Siriporn C. Chattipakorn
Diet-induced obesity, gut microbiota and bone, including alveolar bone loss
description © 2017 Elsevier Ltd Obesity is a major risk factor for several pathologies, including jaw bone resorption. The underlying mechanisms involved in pathological conditions resulting from obesity include chronic systemic inflammation and the development of insulin resistance. Although numerous studies have indicated the importance of the role of gut microbiota in the pathogenesis of inflammation and insulin resistance in obesity, only a few studies have established a relationship between obesity, gut microbiota and status of the jaw bone. This review aims to summarize current findings relating to these issues, focusing on the role of obesity and gut microbiota on jaw bone health, including possible mechanisms which can explain this link.
format Journal
author Sathima Eaimworawuthikul
Parameth Thiennimitr
Nipon Chattipakorn
Siriporn C. Chattipakorn
author_facet Sathima Eaimworawuthikul
Parameth Thiennimitr
Nipon Chattipakorn
Siriporn C. Chattipakorn
author_sort Sathima Eaimworawuthikul
title Diet-induced obesity, gut microbiota and bone, including alveolar bone loss
title_short Diet-induced obesity, gut microbiota and bone, including alveolar bone loss
title_full Diet-induced obesity, gut microbiota and bone, including alveolar bone loss
title_fullStr Diet-induced obesity, gut microbiota and bone, including alveolar bone loss
title_full_unstemmed Diet-induced obesity, gut microbiota and bone, including alveolar bone loss
title_sort diet-induced obesity, gut microbiota and bone, including alveolar bone loss
publishDate 2018
url https://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=85012277820&origin=inward
http://cmuir.cmu.ac.th/jspui/handle/6653943832/46386
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