Inactivation of AKT/NF-κB signaling by eurycomalactone decreases human NSCLC cell viability and improves the chemosensitivity to cisplatin

Inactivation of AKT/NF-κB signaling by eurycomalactone decreases human NSCLC cell viability and improves the chemosensitivity to cisplatin

© This work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International (CC BY-NC-ND 4.0) License. The high activation of protein kinase B (AKT)/nuclear factor-κB (NF-κB) signaling has often been associated with the induction of non-small cell lung cancer (NSCLC) c...

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Main Authors: Nahathai Dukaew, Kongthawat Chairatvit, Pornsiri Pitchakarn, Arisa Imsumran, Jirarat Karinchai, Wirote Tuntiwechapikul, Ariyaphong Wongnoppavich
Format: Journal
Published: 2020
Subjects:
Biochemistry, Genetics and Molecular Biology
Medicine
Online Access:https://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=85089888704&origin=inward
http://cmuir.cmu.ac.th/jspui/handle/6653943832/70165
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Institution: Chiang Mai University

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https://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=85089888704&origin=inward
http://cmuir.cmu.ac.th/jspui/handle/6653943832/70165

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