The chromatin structure of the long control region of human papillomavirus type 16 represses viral oncoprotein expression

The chromatin structure of the long control region of human papillomavirus type 16 represses viral oncoprotein expression

Journal of Virology

محفوظ في:

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Stünkel, W., Bernard, H.-U.
مؤلفون آخرون:	INSTITUTE OF MOLECULAR & CELL BIOLOGY
التنسيق:	مقال
منشور في:	2014
الوصول للمادة أونلاين:	http://scholarbank.nus.edu.sg/handle/10635/112105
الوسوم:	إضافة وسم لا توجد وسوم, كن أول من يضع وسما على هذه التسجيلة!
المؤسسة:	National University of Singapore

مواد مشابهة

Nuclear matrix attachment regions of human papillomavirus type 16 repress or activate the E6 promoter, depending on the physical state of the viral DNA
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YY1 represses human papillomavirus type 16 transcription by quenching AP-1 activity
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Human papillomavirus-16 and genital cancer: are tests for the viral gene expression in vitro indicators for risk factors in vivo?
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Association of human papillomavirus type 16 long control region mutation and cervical cancer
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Association of human papillomavirus type 16 long control region mutation and cervical cancer
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Nuclear matrix attachment regions of human papillomavirus type 16 point toward conservation of these genomic elements in all genital papillomaviruses
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The genetic drift of human papillomavirus type 16 is a means of reconstructing prehistoric viral spread and the movement of ancient human populations
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Numerous nuclear proteins bind the long control region of human papillomavirus type 16: A subset of 6 of 23 DNase I-protected segments coincides with the location of the cell-type-specific enhancer
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The human papillomavirus type 16 E2 transcription factor binds with low cooperativity to two flanking sites and represses the E6 promoter through displacement of Sp1 and TFIID
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Human papillomavirus type 2c is identical to human papillomavirus type 27
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Progesterone and glucocorticoid response elements occur in the long control regions of several human papillomaviruses involved in anogenital neoplasia
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During negative regulation of the human papillomavirus-16 E6 promoter, the viral E2 protein can displace Sp1 from a proximal promoter element
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Human papillomaviruses and cervical cancer in Bangkok. I. Risk factors for invasive cervical carcinomas with human papillomavirus types 16 and 18 DNA
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H3K27me3-rich genomic regions can function as silencers to repress gene expression via chromatin interactions
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H3K27ME3-RICH GENOMIC REGIONS CAN FUNCTION AS SILENCERS TO REPRESS GENE EXPRESSION VIA CHROMATIN INTERACTIONS
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H3K27me3-rich genomic regions can function as silencers to repress gene expression via chromatin interactions
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Coevolution of papillomaviruses with human populations
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The E6/E7 promoter of human papillomavirus type 16 is activated in the absence of E2 proteins by a sequence-aberrant Spl distal element
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Post-Translational Control of IL-1β via the Human Papillomavirus Type 16 E6 Oncoprotein: A Novel Mechanism of Innate Immune Escape Mediated by the E3-Ubiquitin Ligase E6-AP and p53
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Oct-1 activates the epithelial-specific enhancer of human papillomavirus type 16 via a synergistic interaction with NFI at a conserved composite regulatory element
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Sequence variants of human papillomavirus type 16 in clinical samples permit verification and extension of epidemiological studies and construction of a phylogenetic tree
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In vitro and in vivo inhibition of human papillomavirus type 16 E6 and E7 genes
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