Xist RNA repeat E is essential for ASH2L recruitment to the inactive X and regulates histone modifications and escape gene expression

10.1371/journal.pgen.1006890

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Main Authors:	Yue M., Ogawa A., Yamada N., Charles Richard J.L., Barski A., Ogawa Y.
Other Authors:	CANCER SCIENCE INSTITUTE OF SINGAPORE
Format:	Article
Published:	Public Library of Science 2020
Subjects:	long non coding RNA Xist long untranslated RNA membrane protein protein ASH2L unclassified drug Ash2l protein, mouse DNA binding protein nuclear protein transcription factor XIST non-coding RNA animal cell Article chromatin immunoprecipitation controlled study endothelium cell enzyme localization epigenetics exon female gene control gene expression gene inactivation half life time histone modification immunoprecipitation mouse nonhuman protein depletion protein RNA binding quantitative analysis reverse transcription polymerase chain reaction upregulation animal biosynthesis cell differentiation embryonic stem cell gene deletion gene expression regulation gene silencing genetic transcription genetics histone code metabolism X chromosome X chromosome inactivation Animals Cell Differentiation DNA-Binding Proteins Embryonic Stem Cells Exons Gene Deletion Gene Expression Regulation Gene Silencing Histone Code Mice Nuclear Proteins RNA, Long Noncoding Transcription Factors Transcription, Genetic X Chromosome X Chromosome Inactivation
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/165375
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Institution:	National University of Singapore

id	sg-nus-scholar.10635-165375
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spelling	sg-nus-scholar.10635-1653752023-10-31T20:50:11Z Xist RNA repeat E is essential for ASH2L recruitment to the inactive X and regulates histone modifications and escape gene expression Yue M. Ogawa A. Yamada N. Charles Richard J.L. Barski A. Ogawa Y. CANCER SCIENCE INSTITUTE OF SINGAPORE long non coding RNA Xist long untranslated RNA membrane protein protein ASH2L unclassified drug Ash2l protein, mouse DNA binding protein long untranslated RNA nuclear protein transcription factor XIST non-coding RNA animal cell Article chromatin immunoprecipitation controlled study endothelium cell enzyme localization epigenetics exon female gene control gene expression gene inactivation half life time histone modification immunoprecipitation mouse nonhuman protein depletion protein RNA binding quantitative analysis reverse transcription polymerase chain reaction upregulation animal biosynthesis cell differentiation embryonic stem cell gene deletion gene expression regulation gene silencing genetic transcription genetics histone code metabolism X chromosome X chromosome inactivation Animals Cell Differentiation DNA-Binding Proteins Embryonic Stem Cells Exons Gene Deletion Gene Expression Regulation Gene Silencing Histone Code Mice Nuclear Proteins RNA, Long Noncoding Transcription Factors Transcription, Genetic X Chromosome X Chromosome Inactivation 10.1371/journal.pgen.1006890 PLoS Genetics 13 7 e1006890 2020-03-13T05:22:26Z 2020-03-13T05:22:26Z 2017 Article Yue M., Ogawa A., Yamada N., Charles Richard J.L., Barski A., Ogawa Y. (2017). Xist RNA repeat E is essential for ASH2L recruitment to the inactive X and regulates histone modifications and escape gene expression. PLoS Genetics 13 (7) : e1006890. ScholarBank@NUS Repository. https://doi.org/10.1371/journal.pgen.1006890 15537390 https://scholarbank.nus.edu.sg/handle/10635/165375 Public Library of Science Unpaywall 20200320
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	long non coding RNA Xist long untranslated RNA membrane protein protein ASH2L unclassified drug Ash2l protein, mouse DNA binding protein long untranslated RNA nuclear protein transcription factor XIST non-coding RNA animal cell Article chromatin immunoprecipitation controlled study endothelium cell enzyme localization epigenetics exon female gene control gene expression gene inactivation half life time histone modification immunoprecipitation mouse nonhuman protein depletion protein RNA binding quantitative analysis reverse transcription polymerase chain reaction upregulation animal biosynthesis cell differentiation embryonic stem cell gene deletion gene expression regulation gene silencing genetic transcription genetics histone code metabolism X chromosome X chromosome inactivation Animals Cell Differentiation DNA-Binding Proteins Embryonic Stem Cells Exons Gene Deletion Gene Expression Regulation Gene Silencing Histone Code Mice Nuclear Proteins RNA, Long Noncoding Transcription Factors Transcription, Genetic X Chromosome X Chromosome Inactivation
spellingShingle	long non coding RNA Xist long untranslated RNA membrane protein protein ASH2L unclassified drug Ash2l protein, mouse DNA binding protein long untranslated RNA nuclear protein transcription factor XIST non-coding RNA animal cell Article chromatin immunoprecipitation controlled study endothelium cell enzyme localization epigenetics exon female gene control gene expression gene inactivation half life time histone modification immunoprecipitation mouse nonhuman protein depletion protein RNA binding quantitative analysis reverse transcription polymerase chain reaction upregulation animal biosynthesis cell differentiation embryonic stem cell gene deletion gene expression regulation gene silencing genetic transcription genetics histone code metabolism X chromosome X chromosome inactivation Animals Cell Differentiation DNA-Binding Proteins Embryonic Stem Cells Exons Gene Deletion Gene Expression Regulation Gene Silencing Histone Code Mice Nuclear Proteins RNA, Long Noncoding Transcription Factors Transcription, Genetic X Chromosome X Chromosome Inactivation Yue M. Ogawa A. Yamada N. Charles Richard J.L. Barski A. Ogawa Y. Xist RNA repeat E is essential for ASH2L recruitment to the inactive X and regulates histone modifications and escape gene expression
description	10.1371/journal.pgen.1006890
author2	CANCER SCIENCE INSTITUTE OF SINGAPORE
author_facet	CANCER SCIENCE INSTITUTE OF SINGAPORE Yue M. Ogawa A. Yamada N. Charles Richard J.L. Barski A. Ogawa Y.
format	Article
author	Yue M. Ogawa A. Yamada N. Charles Richard J.L. Barski A. Ogawa Y.
author_sort	Yue M.
title	Xist RNA repeat E is essential for ASH2L recruitment to the inactive X and regulates histone modifications and escape gene expression
title_short	Xist RNA repeat E is essential for ASH2L recruitment to the inactive X and regulates histone modifications and escape gene expression
title_full	Xist RNA repeat E is essential for ASH2L recruitment to the inactive X and regulates histone modifications and escape gene expression
title_fullStr	Xist RNA repeat E is essential for ASH2L recruitment to the inactive X and regulates histone modifications and escape gene expression
title_full_unstemmed	Xist RNA repeat E is essential for ASH2L recruitment to the inactive X and regulates histone modifications and escape gene expression
title_sort	xist rna repeat e is essential for ash2l recruitment to the inactive x and regulates histone modifications and escape gene expression
publisher	Public Library of Science
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/165375
_version_	1781791925078065152

Xist RNA repeat E is essential for ASH2L recruitment to the inactive X and regulates histone modifications and escape gene expression

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