Changes in macrophage transcriptome associate with systemic sclerosis and mediate GSDMA contribution to disease risk
10.1136/annrheumdis-2017-212454
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sg-nus-scholar.10635-1751202023-08-29T21:58:16Z Changes in macrophage transcriptome associate with systemic sclerosis and mediate GSDMA contribution to disease risk Moreno-Moral, A Bagnati, M Koturan, S Ko, J.-H Fonseca, C Harmston, N Game, L Martin, J Ong, V Abraham, D.J Denton, C.P Behmoaras, J Petretto, E DUKE-NUS MEDICAL SCHOOL gamma interferon antibody lipopolysaccharide GSDMA protein, human transcriptome tumor protein Article controlled study disease association down regulation epigenetics expression quantitative trait locus gene expression regulation genetic identification genetic risk genetic susceptibility genetic transcription genetic variability genotype glycolysis human hypoxia immune response macrophage major clinical study monocyte mTOR signaling priority journal RNA sequence systemic sclerosis upregulation adolescent adult case control study child cytology female genetic predisposition genetics genotyping technique macrophage male metabolism pathology preschool child quantitative trait locus risk factor signal transduction skin systemic sclerosis young adult Adolescent Adult Case-Control Studies Child Child, Preschool Female Genetic Predisposition to Disease Genotyping Techniques Humans Macrophages Male Neoplasm Proteins Quantitative Trait Loci Risk Factors Scleroderma, Systemic Signal Transduction Skin Transcriptome Young Adult 10.1136/annrheumdis-2017-212454 Annals of the Rheumatic Diseases 77 4 596-601 2020-09-09T04:10:16Z 2020-09-09T04:10:16Z 2018 Article Moreno-Moral, A, Bagnati, M, Koturan, S, Ko, J.-H, Fonseca, C, Harmston, N, Game, L, Martin, J, Ong, V, Abraham, D.J, Denton, C.P, Behmoaras, J, Petretto, E (2018). Changes in macrophage transcriptome associate with systemic sclerosis and mediate GSDMA contribution to disease risk. Annals of the Rheumatic Diseases 77 (4) : 596-601. ScholarBank@NUS Repository. https://doi.org/10.1136/annrheumdis-2017-212454 0003-4967 https://scholarbank.nus.edu.sg/handle/10635/175120 BMJ Publishing Group Unpaywall 20200831 |
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gamma interferon antibody lipopolysaccharide GSDMA protein, human transcriptome tumor protein Article controlled study disease association down regulation epigenetics expression quantitative trait locus gene expression regulation genetic identification genetic risk genetic susceptibility genetic transcription genetic variability genotype glycolysis human hypoxia immune response macrophage major clinical study monocyte mTOR signaling priority journal RNA sequence systemic sclerosis upregulation adolescent adult case control study child cytology female genetic predisposition genetics genotyping technique macrophage male metabolism pathology preschool child quantitative trait locus risk factor signal transduction skin systemic sclerosis young adult Adolescent Adult Case-Control Studies Child Child, Preschool Female Genetic Predisposition to Disease Genotyping Techniques Humans Macrophages Male Neoplasm Proteins Quantitative Trait Loci Risk Factors Scleroderma, Systemic Signal Transduction Skin Transcriptome Young Adult |
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gamma interferon antibody lipopolysaccharide GSDMA protein, human transcriptome tumor protein Article controlled study disease association down regulation epigenetics expression quantitative trait locus gene expression regulation genetic identification genetic risk genetic susceptibility genetic transcription genetic variability genotype glycolysis human hypoxia immune response macrophage major clinical study monocyte mTOR signaling priority journal RNA sequence systemic sclerosis upregulation adolescent adult case control study child cytology female genetic predisposition genetics genotyping technique macrophage male metabolism pathology preschool child quantitative trait locus risk factor signal transduction skin systemic sclerosis young adult Adolescent Adult Case-Control Studies Child Child, Preschool Female Genetic Predisposition to Disease Genotyping Techniques Humans Macrophages Male Neoplasm Proteins Quantitative Trait Loci Risk Factors Scleroderma, Systemic Signal Transduction Skin Transcriptome Young Adult Moreno-Moral, A Bagnati, M Koturan, S Ko, J.-H Fonseca, C Harmston, N Game, L Martin, J Ong, V Abraham, D.J Denton, C.P Behmoaras, J Petretto, E Changes in macrophage transcriptome associate with systemic sclerosis and mediate GSDMA contribution to disease risk |
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10.1136/annrheumdis-2017-212454 |
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DUKE-NUS MEDICAL SCHOOL |
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DUKE-NUS MEDICAL SCHOOL Moreno-Moral, A Bagnati, M Koturan, S Ko, J.-H Fonseca, C Harmston, N Game, L Martin, J Ong, V Abraham, D.J Denton, C.P Behmoaras, J Petretto, E |
format |
Article |
author |
Moreno-Moral, A Bagnati, M Koturan, S Ko, J.-H Fonseca, C Harmston, N Game, L Martin, J Ong, V Abraham, D.J Denton, C.P Behmoaras, J Petretto, E |
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Moreno-Moral, A |
title |
Changes in macrophage transcriptome associate with systemic sclerosis and mediate GSDMA contribution to disease risk |
title_short |
Changes in macrophage transcriptome associate with systemic sclerosis and mediate GSDMA contribution to disease risk |
title_full |
Changes in macrophage transcriptome associate with systemic sclerosis and mediate GSDMA contribution to disease risk |
title_fullStr |
Changes in macrophage transcriptome associate with systemic sclerosis and mediate GSDMA contribution to disease risk |
title_full_unstemmed |
Changes in macrophage transcriptome associate with systemic sclerosis and mediate GSDMA contribution to disease risk |
title_sort |
changes in macrophage transcriptome associate with systemic sclerosis and mediate gsdma contribution to disease risk |
publisher |
BMJ Publishing Group |
publishDate |
2020 |
url |
https://scholarbank.nus.edu.sg/handle/10635/175120 |
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1778169856136314880 |