TCR-like antibodies mediate complement and antibody-dependent cellular cytotoxicity against Epstein-Barr virus-transformed B lymphoblastoid cells expressing different HLA-A?02 microvariants

• Main Authors: Lai J., Choo J.A.L., Tan W.J., Too C.T., Oo M.Z., Suter M.A., Mustafa F.B., Srinivasan N., Chan C.E.Z., Lim A.G.X., Zhong Y., Chan S.H., Hanson B.J., Gascoigne N.R.J., MacAry P.A.
• Other Authors: MEDICINE
• Format: Article
• Published: 2020
• Subjects: HLA A2 antigen; HLA-A*02 antigen; lymphocyte antigen receptor; monoclonal antibody; peptide fragment; antibody dependent cellular cytotoxicity; chemistry; Epstein Barr virus; genetic variation; genetics; herpes virus infection; human; immunology; metabolism; molecular model; tumor cell line; Antibodies, Monoclonal; Antibody-Dependent Cell Cytotoxicity; Cell Line, Tumor; Genetic Variation; Herpesviridae Infections; Herpesvirus 4, Human; HLA-A2 Antigen; Humans; Models, Molecular; Peptide Fragments; Receptors, Antigen, T-Cell
• Online Access: https://scholarbank.nus.edu.sg/handle/10635/175175
• Institution: National University of Singapore

10.1038/s41598-017-10265-6