Nrf2 is a central regulator of metabolic reprogramming of myeloid-derived suppressor cells in steady state and sepsis
10.3389/fimmu.2018.01552
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sg-nus-scholar.10635-1780792023-10-31T21:26:18Z Nrf2 is a central regulator of metabolic reprogramming of myeloid-derived suppressor cells in steady state and sepsis Ohl, K Fragoulis, A Klemm, P Baumeister, J Klock, W Verjans, E Böll, S Möllmann, J Lehrke, M Costa, I Denecke, B Schippers, A Roth, J Wagner, N Wruck, C Tenbrock, K SAW SWEE HOCK SCHOOL OF PUBLIC HEALTH CD11b antigen CD4 antigen complementary DNA deoxyribonuclease I granulocyte macrophage colony stimulating factor inducible nitric oxide synthase interleukin 1 interleukin 12 interleukin 6 kelch like ECH associated protein 1 toll like receptor 4 transcription factor Nrf2 transcriptome uracil DNA glycosidase aerobic glycolysis animal cell animal experiment animal model animal tissue Article CD4+ T lymphocyte cell cycle cell expansion colitis controlled study gene expression immunological tolerance immunomodulation in vitro study in vivo study lipopolysaccharide-induced sepsis metabolic activation metabolic regulation mitochondrial respiration mouse myeloid-derived suppressor cell nonhuman nuclear reprogramming oxidative stress transcriptomics 10.3389/fimmu.2018.01552 Frontiers in Immunology 9 JUL 1552 2020-10-20T05:02:00Z 2020-10-20T05:02:00Z 2018 Article Ohl, K, Fragoulis, A, Klemm, P, Baumeister, J, Klock, W, Verjans, E, Böll, S, Möllmann, J, Lehrke, M, Costa, I, Denecke, B, Schippers, A, Roth, J, Wagner, N, Wruck, C, Tenbrock, K (2018). Nrf2 is a central regulator of metabolic reprogramming of myeloid-derived suppressor cells in steady state and sepsis. Frontiers in Immunology 9 (JUL) : 1552. ScholarBank@NUS Repository. https://doi.org/10.3389/fimmu.2018.01552 16643224 https://scholarbank.nus.edu.sg/handle/10635/178079 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ Unpaywall 20201031 |
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Singapore Singapore |
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CD11b antigen CD4 antigen complementary DNA deoxyribonuclease I granulocyte macrophage colony stimulating factor inducible nitric oxide synthase interleukin 1 interleukin 12 interleukin 6 kelch like ECH associated protein 1 toll like receptor 4 transcription factor Nrf2 transcriptome uracil DNA glycosidase aerobic glycolysis animal cell animal experiment animal model animal tissue Article CD4+ T lymphocyte cell cycle cell expansion colitis controlled study gene expression immunological tolerance immunomodulation in vitro study in vivo study lipopolysaccharide-induced sepsis metabolic activation metabolic regulation mitochondrial respiration mouse myeloid-derived suppressor cell nonhuman nuclear reprogramming oxidative stress transcriptomics |
| spellingShingle |
CD11b antigen CD4 antigen complementary DNA deoxyribonuclease I granulocyte macrophage colony stimulating factor inducible nitric oxide synthase interleukin 1 interleukin 12 interleukin 6 kelch like ECH associated protein 1 toll like receptor 4 transcription factor Nrf2 transcriptome uracil DNA glycosidase aerobic glycolysis animal cell animal experiment animal model animal tissue Article CD4+ T lymphocyte cell cycle cell expansion colitis controlled study gene expression immunological tolerance immunomodulation in vitro study in vivo study lipopolysaccharide-induced sepsis metabolic activation metabolic regulation mitochondrial respiration mouse myeloid-derived suppressor cell nonhuman nuclear reprogramming oxidative stress transcriptomics Ohl, K Fragoulis, A Klemm, P Baumeister, J Klock, W Verjans, E Böll, S Möllmann, J Lehrke, M Costa, I Denecke, B Schippers, A Roth, J Wagner, N Wruck, C Tenbrock, K Nrf2 is a central regulator of metabolic reprogramming of myeloid-derived suppressor cells in steady state and sepsis |
| description |
10.3389/fimmu.2018.01552 |
| author2 |
SAW SWEE HOCK SCHOOL OF PUBLIC HEALTH |
| author_facet |
SAW SWEE HOCK SCHOOL OF PUBLIC HEALTH Ohl, K Fragoulis, A Klemm, P Baumeister, J Klock, W Verjans, E Böll, S Möllmann, J Lehrke, M Costa, I Denecke, B Schippers, A Roth, J Wagner, N Wruck, C Tenbrock, K |
| format |
Article |
| author |
Ohl, K Fragoulis, A Klemm, P Baumeister, J Klock, W Verjans, E Böll, S Möllmann, J Lehrke, M Costa, I Denecke, B Schippers, A Roth, J Wagner, N Wruck, C Tenbrock, K |
| author_sort |
Ohl, K |
| title |
Nrf2 is a central regulator of metabolic reprogramming of myeloid-derived suppressor cells in steady state and sepsis |
| title_short |
Nrf2 is a central regulator of metabolic reprogramming of myeloid-derived suppressor cells in steady state and sepsis |
| title_full |
Nrf2 is a central regulator of metabolic reprogramming of myeloid-derived suppressor cells in steady state and sepsis |
| title_fullStr |
Nrf2 is a central regulator of metabolic reprogramming of myeloid-derived suppressor cells in steady state and sepsis |
| title_full_unstemmed |
Nrf2 is a central regulator of metabolic reprogramming of myeloid-derived suppressor cells in steady state and sepsis |
| title_sort |
nrf2 is a central regulator of metabolic reprogramming of myeloid-derived suppressor cells in steady state and sepsis |
| publishDate |
2020 |
| url |
https://scholarbank.nus.edu.sg/handle/10635/178079 |
| _version_ |
1781792275736559616 |