Suppression of TAK1 pathway by shear stress counteracts the inflammatory endothelial cell phenotype induced by oxidative stress and TGF-?1

10.1038/srep42487

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Main Authors:	Lee, E.S, Boldo, L.S, Fernandez, B.O, Feelisch, M, Harmsen, M.C
Other Authors:	PHYSIOLOGY
Format:	Article
Published:	2020
Subjects:	autacoid biological marker immunoglobulin enhancer binding protein MAP kinase kinase kinase 7 mitogen activated protein kinase 7 mitogen activated protein kinase kinase kinase reactive oxygen metabolite transforming growth factor beta1 animal biological model drug effect endothelium cell genetics human metabolism mitochondrion oxidative stress phenotype rat shear strength signal transduction umbilical vein endothelial cell Animals Biomarkers Endothelial Cells Human Umbilical Vein Endothelial Cells Humans Inflammation Mediators MAP Kinase Kinase Kinases Mitochondria Mitogen-Activated Protein Kinase 7 Models, Biological NF-kappa B Oxidative Stress Phenotype Rats Reactive Oxygen Species Shear Strength Signal Transduction Transforming Growth Factor beta1
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/178699
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Institution:	National University of Singapore

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spelling	sg-nus-scholar.10635-1786992024-04-04T01:07:23Z Suppression of TAK1 pathway by shear stress counteracts the inflammatory endothelial cell phenotype induced by oxidative stress and TGF-?1 Lee, E.S Boldo, L.S Fernandez, B.O Feelisch, M Harmsen, M.C PHYSIOLOGY autacoid biological marker immunoglobulin enhancer binding protein MAP kinase kinase kinase 7 mitogen activated protein kinase 7 mitogen activated protein kinase kinase kinase reactive oxygen metabolite transforming growth factor beta1 animal biological model drug effect endothelium cell genetics human metabolism mitochondrion oxidative stress phenotype rat shear strength signal transduction umbilical vein endothelial cell Animals Biomarkers Endothelial Cells Human Umbilical Vein Endothelial Cells Humans Inflammation Mediators MAP Kinase Kinase Kinases Mitochondria Mitogen-Activated Protein Kinase 7 Models, Biological NF-kappa B Oxidative Stress Phenotype Rats Reactive Oxygen Species Shear Strength Signal Transduction Transforming Growth Factor beta1 10.1038/srep42487 Scientific Reports 7 42487 2020-10-21T07:59:13Z 2020-10-21T07:59:13Z 2017 Article Lee, E.S, Boldo, L.S, Fernandez, B.O, Feelisch, M, Harmsen, M.C (2017). Suppression of TAK1 pathway by shear stress counteracts the inflammatory endothelial cell phenotype induced by oxidative stress and TGF-?1. Scientific Reports 7 : 42487. ScholarBank@NUS Repository. https://doi.org/10.1038/srep42487 20452322 https://scholarbank.nus.edu.sg/handle/10635/178699 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ Unpaywall 20201031
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	autacoid biological marker immunoglobulin enhancer binding protein MAP kinase kinase kinase 7 mitogen activated protein kinase 7 mitogen activated protein kinase kinase kinase reactive oxygen metabolite transforming growth factor beta1 animal biological model drug effect endothelium cell genetics human metabolism mitochondrion oxidative stress phenotype rat shear strength signal transduction umbilical vein endothelial cell Animals Biomarkers Endothelial Cells Human Umbilical Vein Endothelial Cells Humans Inflammation Mediators MAP Kinase Kinase Kinases Mitochondria Mitogen-Activated Protein Kinase 7 Models, Biological NF-kappa B Oxidative Stress Phenotype Rats Reactive Oxygen Species Shear Strength Signal Transduction Transforming Growth Factor beta1
spellingShingle	autacoid biological marker immunoglobulin enhancer binding protein MAP kinase kinase kinase 7 mitogen activated protein kinase 7 mitogen activated protein kinase kinase kinase reactive oxygen metabolite transforming growth factor beta1 animal biological model drug effect endothelium cell genetics human metabolism mitochondrion oxidative stress phenotype rat shear strength signal transduction umbilical vein endothelial cell Animals Biomarkers Endothelial Cells Human Umbilical Vein Endothelial Cells Humans Inflammation Mediators MAP Kinase Kinase Kinases Mitochondria Mitogen-Activated Protein Kinase 7 Models, Biological NF-kappa B Oxidative Stress Phenotype Rats Reactive Oxygen Species Shear Strength Signal Transduction Transforming Growth Factor beta1 Lee, E.S Boldo, L.S Fernandez, B.O Feelisch, M Harmsen, M.C Suppression of TAK1 pathway by shear stress counteracts the inflammatory endothelial cell phenotype induced by oxidative stress and TGF-?1
description	10.1038/srep42487
author2	PHYSIOLOGY
author_facet	PHYSIOLOGY Lee, E.S Boldo, L.S Fernandez, B.O Feelisch, M Harmsen, M.C
format	Article
author	Lee, E.S Boldo, L.S Fernandez, B.O Feelisch, M Harmsen, M.C
author_sort	Lee, E.S
title	Suppression of TAK1 pathway by shear stress counteracts the inflammatory endothelial cell phenotype induced by oxidative stress and TGF-?1
title_short	Suppression of TAK1 pathway by shear stress counteracts the inflammatory endothelial cell phenotype induced by oxidative stress and TGF-?1
title_full	Suppression of TAK1 pathway by shear stress counteracts the inflammatory endothelial cell phenotype induced by oxidative stress and TGF-?1
title_fullStr	Suppression of TAK1 pathway by shear stress counteracts the inflammatory endothelial cell phenotype induced by oxidative stress and TGF-?1
title_full_unstemmed	Suppression of TAK1 pathway by shear stress counteracts the inflammatory endothelial cell phenotype induced by oxidative stress and TGF-?1
title_sort	suppression of tak1 pathway by shear stress counteracts the inflammatory endothelial cell phenotype induced by oxidative stress and tgf-?1
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/178699
_version_	1795374467239641088

Suppression of TAK1 pathway by shear stress counteracts the inflammatory endothelial cell phenotype induced by oxidative stress and TGF-?1

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