Mice deleted for cell division cycle 73 gene develop parathyroid and uterine tumours: Model for the hyperparathyroidism-jaw tumour syndrome

• Main Authors: Walls, G.V, Stevenson, M, Lines, K.E, Newey, P.J, Reed, A.A.C, Bowl, M.R, Jeyabalan, J, Harding, B, Bradley, K.J, Manek, S, Chen, J, Wang, P, Williams, B.O, Teh, B.T, Thakker, R.V
• Other Authors: DUKE-NUS MEDICAL SCHOOL
• Format: Article
• Published: Nature Publishing Group 2020
• Subjects: calcium; galectin 3; parathyroid hormone; progesterone receptor; HRPT2 protein, mouse; tumor suppressor protein; adenofibroma; adenomyoma; allele; animal cell; animal experiment; animal tissue; Article; binding site; calcium blood level; carcinogenesis; cell division; cell proliferation; cell survival; controlled study; crossing over; exon; female; hyperparathyroidism; in vitro study; in vivo study; jaw tumor; male; mammal cell; mouse; nonhuman; parathyroid adenoma; parathyroid gland; parathyroid hormone blood level; priority journal; protein expression; squamous cell metaplasia; stop codon; tumor syndrome; uterus cancer; uterus tissue; Western blotting; wild type; adenoma; animal; C57BL mouse; carcinoma; complication; fibroma; gene deletion; genetics; knockout mouse; parathyroid tumor; Adenoma; Animals; Carcinoma; Female; Fibroma; Gene Deletion; Hyperparathyroidism; Jaw Neoplasms; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Parathyroid Neoplasms; Tumor Suppressor Proteins; Uterine Neoplasms
• Online Access: https://scholarbank.nus.edu.sg/handle/10635/179192
• Institution: National University of Singapore

10.1038/onc.2017.43