Mice deleted for cell division cycle 73 gene develop parathyroid and uterine tumours: Model for the hyperparathyroidism-jaw tumour syndrome

10.1038/onc.2017.43

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Main Authors:	Walls, G.V, Stevenson, M, Lines, K.E, Newey, P.J, Reed, A.A.C, Bowl, M.R, Jeyabalan, J, Harding, B, Bradley, K.J, Manek, S, Chen, J, Wang, P, Williams, B.O, Teh, B.T, Thakker, R.V
Other Authors:	DUKE-NUS MEDICAL SCHOOL
Format:	Article
Published:	Nature Publishing Group 2020
Subjects:	calcium galectin 3 parathyroid hormone progesterone receptor HRPT2 protein, mouse tumor suppressor protein adenofibroma adenomyoma allele animal cell animal experiment animal tissue Article binding site calcium blood level carcinogenesis cell division cell proliferation cell survival controlled study crossing over exon female hyperparathyroidism in vitro study in vivo study jaw tumor male mammal cell mouse nonhuman parathyroid adenoma parathyroid gland parathyroid hormone blood level priority journal protein expression squamous cell metaplasia stop codon tumor syndrome uterus cancer uterus tissue Western blotting wild type adenoma animal C57BL mouse carcinoma complication fibroma gene deletion genetics knockout mouse parathyroid tumor Adenoma Animals Carcinoma Female Fibroma Gene Deletion Hyperparathyroidism Jaw Neoplasms Male Mice Mice, Inbred C57BL Mice, Knockout Parathyroid Neoplasms Tumor Suppressor Proteins Uterine Neoplasms
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/179192
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Institution:	National University of Singapore

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spelling	sg-nus-scholar.10635-1791922024-11-09T02:38:35Z Mice deleted for cell division cycle 73 gene develop parathyroid and uterine tumours: Model for the hyperparathyroidism-jaw tumour syndrome Walls, G.V Stevenson, M Lines, K.E Newey, P.J Reed, A.A.C Bowl, M.R Jeyabalan, J Harding, B Bradley, K.J Manek, S Chen, J Wang, P Williams, B.O Teh, B.T Thakker, R.V DUKE-NUS MEDICAL SCHOOL calcium galectin 3 parathyroid hormone progesterone receptor HRPT2 protein, mouse tumor suppressor protein adenofibroma adenomyoma allele animal cell animal experiment animal tissue Article binding site calcium blood level carcinogenesis cell division cell proliferation cell survival controlled study crossing over exon female hyperparathyroidism in vitro study in vivo study jaw tumor male mammal cell mouse nonhuman parathyroid adenoma parathyroid gland parathyroid hormone blood level priority journal protein expression squamous cell metaplasia stop codon tumor syndrome uterus cancer uterus tissue Western blotting wild type adenoma animal C57BL mouse carcinoma complication fibroma gene deletion genetics hyperparathyroidism jaw tumor knockout mouse parathyroid tumor uterus cancer Adenoma Animals Carcinoma Female Fibroma Gene Deletion Hyperparathyroidism Jaw Neoplasms Male Mice Mice, Inbred C57BL Mice, Knockout Parathyroid Neoplasms Tumor Suppressor Proteins Uterine Neoplasms 10.1038/onc.2017.43 Oncogene 36 28 4025-4036 2020-10-23T02:27:01Z 2020-10-23T02:27:01Z 2017 Article Walls, G.V, Stevenson, M, Lines, K.E, Newey, P.J, Reed, A.A.C, Bowl, M.R, Jeyabalan, J, Harding, B, Bradley, K.J, Manek, S, Chen, J, Wang, P, Williams, B.O, Teh, B.T, Thakker, R.V (2017). Mice deleted for cell division cycle 73 gene develop parathyroid and uterine tumours: Model for the hyperparathyroidism-jaw tumour syndrome. Oncogene 36 (28) : 4025-4036. ScholarBank@NUS Repository. https://doi.org/10.1038/onc.2017.43 09509232 https://scholarbank.nus.edu.sg/handle/10635/179192 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ Nature Publishing Group Unpaywall 20201031
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	calcium galectin 3 parathyroid hormone progesterone receptor HRPT2 protein, mouse tumor suppressor protein adenofibroma adenomyoma allele animal cell animal experiment animal tissue Article binding site calcium blood level carcinogenesis cell division cell proliferation cell survival controlled study crossing over exon female hyperparathyroidism in vitro study in vivo study jaw tumor male mammal cell mouse nonhuman parathyroid adenoma parathyroid gland parathyroid hormone blood level priority journal protein expression squamous cell metaplasia stop codon tumor syndrome uterus cancer uterus tissue Western blotting wild type adenoma animal C57BL mouse carcinoma complication fibroma gene deletion genetics hyperparathyroidism jaw tumor knockout mouse parathyroid tumor uterus cancer Adenoma Animals Carcinoma Female Fibroma Gene Deletion Hyperparathyroidism Jaw Neoplasms Male Mice Mice, Inbred C57BL Mice, Knockout Parathyroid Neoplasms Tumor Suppressor Proteins Uterine Neoplasms
spellingShingle	calcium galectin 3 parathyroid hormone progesterone receptor HRPT2 protein, mouse tumor suppressor protein adenofibroma adenomyoma allele animal cell animal experiment animal tissue Article binding site calcium blood level carcinogenesis cell division cell proliferation cell survival controlled study crossing over exon female hyperparathyroidism in vitro study in vivo study jaw tumor male mammal cell mouse nonhuman parathyroid adenoma parathyroid gland parathyroid hormone blood level priority journal protein expression squamous cell metaplasia stop codon tumor syndrome uterus cancer uterus tissue Western blotting wild type adenoma animal C57BL mouse carcinoma complication fibroma gene deletion genetics hyperparathyroidism jaw tumor knockout mouse parathyroid tumor uterus cancer Adenoma Animals Carcinoma Female Fibroma Gene Deletion Hyperparathyroidism Jaw Neoplasms Male Mice Mice, Inbred C57BL Mice, Knockout Parathyroid Neoplasms Tumor Suppressor Proteins Uterine Neoplasms Walls, G.V Stevenson, M Lines, K.E Newey, P.J Reed, A.A.C Bowl, M.R Jeyabalan, J Harding, B Bradley, K.J Manek, S Chen, J Wang, P Williams, B.O Teh, B.T Thakker, R.V Mice deleted for cell division cycle 73 gene develop parathyroid and uterine tumours: Model for the hyperparathyroidism-jaw tumour syndrome
description	10.1038/onc.2017.43
author2	DUKE-NUS MEDICAL SCHOOL
author_facet	DUKE-NUS MEDICAL SCHOOL Walls, G.V Stevenson, M Lines, K.E Newey, P.J Reed, A.A.C Bowl, M.R Jeyabalan, J Harding, B Bradley, K.J Manek, S Chen, J Wang, P Williams, B.O Teh, B.T Thakker, R.V
format	Article
author	Walls, G.V Stevenson, M Lines, K.E Newey, P.J Reed, A.A.C Bowl, M.R Jeyabalan, J Harding, B Bradley, K.J Manek, S Chen, J Wang, P Williams, B.O Teh, B.T Thakker, R.V
author_sort	Walls, G.V
title	Mice deleted for cell division cycle 73 gene develop parathyroid and uterine tumours: Model for the hyperparathyroidism-jaw tumour syndrome
title_short	Mice deleted for cell division cycle 73 gene develop parathyroid and uterine tumours: Model for the hyperparathyroidism-jaw tumour syndrome
title_full	Mice deleted for cell division cycle 73 gene develop parathyroid and uterine tumours: Model for the hyperparathyroidism-jaw tumour syndrome
title_fullStr	Mice deleted for cell division cycle 73 gene develop parathyroid and uterine tumours: Model for the hyperparathyroidism-jaw tumour syndrome
title_full_unstemmed	Mice deleted for cell division cycle 73 gene develop parathyroid and uterine tumours: Model for the hyperparathyroidism-jaw tumour syndrome
title_sort	mice deleted for cell division cycle 73 gene develop parathyroid and uterine tumours: model for the hyperparathyroidism-jaw tumour syndrome
publisher	Nature Publishing Group
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/179192
_version_	1821200930169683968

Mice deleted for cell division cycle 73 gene develop parathyroid and uterine tumours: Model for the hyperparathyroidism-jaw tumour syndrome

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