Genes from a translational analysis support a multifactorial nature of white matter hyperintensities

10.1161/STROKEAHA.114.007649

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Main Authors:	Lopez, L.M, Hill, W.D, Harris, S.E, Valdes Hernandez, M, Munoz Maniega, S, Bastin, M.E, Bailey, E, Smith, C, McBride, M, McClure, J, Graham, D, Dominiczak, A, Yang, Q, Fornage, M, Ikram, M.A, Debette, S, Launer, L, Bis, J.C, Schmidt, R, Seshadri, S, Porteous, D.J, Starr, J, Deary, I.J, Wardlaw, J.M
Other Authors:	DUKE-NUS MEDICAL SCHOOL
Format:	Article
Published:	Lippincott Williams and Wilkins 2020
Subjects:	nuclear receptor NR4A3 afp gene alb gene Article brain size chromosome 1 chromosome 10 chromosome 13 chromosome 17 farp1 gene gene gene expression genetic association gnai1 gene human inpp5d gene major clinical study mrpl18 gene nonhuman priority journal rat rbm8a gene single nucleotide polymorphism sipa1l2 gene stroke prone spontaneously hypertensive rat white matter white matter hyperintensity xpnpep1 gene aged Alzheimer disease animal brain causality dementia female genetics genome-wide association study Leukoencephalopathies male pathology procedures risk factor spontaneously hypertensive rat translational research Wistar rat Aged Alzheimer Disease Animals Brain Causality Dementia Female Genome-Wide Association Study Humans Male Polymorphism, Single Nucleotide Rats Rats, Inbred SHR Rats, Wistar Risk Factors Translational Medical Research White Matter
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/180128
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Institution:	National University of Singapore

id	sg-nus-scholar.10635-180128
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spelling	sg-nus-scholar.10635-1801282023-11-01T08:46:34Z Genes from a translational analysis support a multifactorial nature of white matter hyperintensities Lopez, L.M Hill, W.D Harris, S.E Valdes Hernandez, M Munoz Maniega, S Bastin, M.E Bailey, E Smith, C McBride, M McClure, J Graham, D Dominiczak, A Yang, Q Fornage, M Ikram, M.A Debette, S Launer, L Bis, J.C Schmidt, R Seshadri, S Porteous, D.J Starr, J Deary, I.J Wardlaw, J.M DUKE-NUS MEDICAL SCHOOL nuclear receptor NR4A3 afp gene alb gene Article brain size chromosome 1 chromosome 10 chromosome 13 chromosome 17 farp1 gene gene gene expression genetic association gnai1 gene human inpp5d gene major clinical study mrpl18 gene nonhuman priority journal rat rbm8a gene single nucleotide polymorphism sipa1l2 gene stroke prone spontaneously hypertensive rat white matter white matter hyperintensity xpnpep1 gene aged Alzheimer disease animal brain causality dementia female genetics genome-wide association study Leukoencephalopathies male pathology procedures risk factor spontaneously hypertensive rat translational research white matter Wistar rat Aged Alzheimer Disease Animals Brain Causality Dementia Female Genome-Wide Association Study Humans Leukoencephalopathies Male Polymorphism, Single Nucleotide Rats Rats, Inbred SHR Rats, Wistar Risk Factors Translational Medical Research White Matter 10.1161/STROKEAHA.114.007649 Stroke 46 2 341-347 2020-10-26T07:06:51Z 2020-10-26T07:06:51Z 2015 Article Lopez, L.M, Hill, W.D, Harris, S.E, Valdes Hernandez, M, Munoz Maniega, S, Bastin, M.E, Bailey, E, Smith, C, McBride, M, McClure, J, Graham, D, Dominiczak, A, Yang, Q, Fornage, M, Ikram, M.A, Debette, S, Launer, L, Bis, J.C, Schmidt, R, Seshadri, S, Porteous, D.J, Starr, J, Deary, I.J, Wardlaw, J.M (2015). Genes from a translational analysis support a multifactorial nature of white matter hyperintensities. Stroke 46 (2) : 341-347. ScholarBank@NUS Repository. https://doi.org/10.1161/STROKEAHA.114.007649 0039-2499 https://scholarbank.nus.edu.sg/handle/10635/180128 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ Lippincott Williams and Wilkins Unpaywall 20201031
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	nuclear receptor NR4A3 afp gene alb gene Article brain size chromosome 1 chromosome 10 chromosome 13 chromosome 17 farp1 gene gene gene expression genetic association gnai1 gene human inpp5d gene major clinical study mrpl18 gene nonhuman priority journal rat rbm8a gene single nucleotide polymorphism sipa1l2 gene stroke prone spontaneously hypertensive rat white matter white matter hyperintensity xpnpep1 gene aged Alzheimer disease animal brain causality dementia female genetics genome-wide association study Leukoencephalopathies male pathology procedures risk factor spontaneously hypertensive rat translational research white matter Wistar rat Aged Alzheimer Disease Animals Brain Causality Dementia Female Genome-Wide Association Study Humans Leukoencephalopathies Male Polymorphism, Single Nucleotide Rats Rats, Inbred SHR Rats, Wistar Risk Factors Translational Medical Research White Matter
spellingShingle	nuclear receptor NR4A3 afp gene alb gene Article brain size chromosome 1 chromosome 10 chromosome 13 chromosome 17 farp1 gene gene gene expression genetic association gnai1 gene human inpp5d gene major clinical study mrpl18 gene nonhuman priority journal rat rbm8a gene single nucleotide polymorphism sipa1l2 gene stroke prone spontaneously hypertensive rat white matter white matter hyperintensity xpnpep1 gene aged Alzheimer disease animal brain causality dementia female genetics genome-wide association study Leukoencephalopathies male pathology procedures risk factor spontaneously hypertensive rat translational research white matter Wistar rat Aged Alzheimer Disease Animals Brain Causality Dementia Female Genome-Wide Association Study Humans Leukoencephalopathies Male Polymorphism, Single Nucleotide Rats Rats, Inbred SHR Rats, Wistar Risk Factors Translational Medical Research White Matter Lopez, L.M Hill, W.D Harris, S.E Valdes Hernandez, M Munoz Maniega, S Bastin, M.E Bailey, E Smith, C McBride, M McClure, J Graham, D Dominiczak, A Yang, Q Fornage, M Ikram, M.A Debette, S Launer, L Bis, J.C Schmidt, R Seshadri, S Porteous, D.J Starr, J Deary, I.J Wardlaw, J.M Genes from a translational analysis support a multifactorial nature of white matter hyperintensities
description	10.1161/STROKEAHA.114.007649
author2	DUKE-NUS MEDICAL SCHOOL
author_facet	DUKE-NUS MEDICAL SCHOOL Lopez, L.M Hill, W.D Harris, S.E Valdes Hernandez, M Munoz Maniega, S Bastin, M.E Bailey, E Smith, C McBride, M McClure, J Graham, D Dominiczak, A Yang, Q Fornage, M Ikram, M.A Debette, S Launer, L Bis, J.C Schmidt, R Seshadri, S Porteous, D.J Starr, J Deary, I.J Wardlaw, J.M
format	Article
author	Lopez, L.M Hill, W.D Harris, S.E Valdes Hernandez, M Munoz Maniega, S Bastin, M.E Bailey, E Smith, C McBride, M McClure, J Graham, D Dominiczak, A Yang, Q Fornage, M Ikram, M.A Debette, S Launer, L Bis, J.C Schmidt, R Seshadri, S Porteous, D.J Starr, J Deary, I.J Wardlaw, J.M
author_sort	Lopez, L.M
title	Genes from a translational analysis support a multifactorial nature of white matter hyperintensities
title_short	Genes from a translational analysis support a multifactorial nature of white matter hyperintensities
title_full	Genes from a translational analysis support a multifactorial nature of white matter hyperintensities
title_fullStr	Genes from a translational analysis support a multifactorial nature of white matter hyperintensities
title_full_unstemmed	Genes from a translational analysis support a multifactorial nature of white matter hyperintensities
title_sort	genes from a translational analysis support a multifactorial nature of white matter hyperintensities
publisher	Lippincott Williams and Wilkins
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/180128
_version_	1781792365708574720

Genes from a translational analysis support a multifactorial nature of white matter hyperintensities

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