Maelstrom promotes hepatocellular carcinoma metastasis by inducing epithelial-mesenchymal transition by way of Akt/GSK-3β/Snail signaling

10.1002/hep.26677

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Main Authors:	Liu, L, Dai, Y, Chen, J, Zeng, T, Li, Y, Chen, L, Zhu, Y.-H, Li, J, Ma, S, Xie, D, Yuan, Y.-F, Guan, X.-Y
Other Authors:	ANATOMY
Format:	Article
Published:	John Wiley and Sons Inc. 2020
Subjects:	activated leukocyte cell adhesion molecule alpha catenin beta catenin BMI1 protein CD133 antigen CD24 antigen cell surface marker endoglin fibronectin glycogen synthase kinase 3beta Hermes antigen membrane antigen messenger RNA Notch1 receptor octamer transcription factor 4 protein kinase B short hairpin RNA transcription factor NANOG transcription factor Snail uvomorulin vimentin adult animal experiment animal model animal tissue article cancer recurrence cancer resistance cancer stem cell cancer survival carcinogenesis carcinogenicity cell growth cell migration cell motility cell renewal chemosensitivity chromosome 1q controlled study enzyme activity epithelial mesenchymal transition female gene amplification gene overexpression gene silencing human human cell human tissue liver cell carcinoma Maelstrom gene male metastasis middle aged mouse multidrug resistance nonhuman oncogene overall survival priority journal RNA interference signal transduction tumor invasion tumor volume tumor xenograft upregulation Animals Carcinoma, Hepatocellular Carrier Proteins Cell Movement Cell Proliferation Disease Models, Animal Disease Progression Epithelial-Mesenchymal Transition Female Glycogen Synthase Kinase 3 Humans Liver Neoplasms Male Mice Middle Aged Neoplasm Metastasis Proto-Oncogene Proteins c-akt RNA, Messenger Signal Transduction Transcription Factors Up-Regulation
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/180768
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Institution:	National University of Singapore

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spelling	sg-nus-scholar.10635-1807682024-11-09T05:04:12Z Maelstrom promotes hepatocellular carcinoma metastasis by inducing epithelial-mesenchymal transition by way of Akt/GSK-3β/Snail signaling Liu, L Dai, Y Chen, J Zeng, T Li, Y Chen, L Zhu, Y.-H Li, J Li, Y Ma, S Xie, D Yuan, Y.-F Guan, X.-Y ANATOMY activated leukocyte cell adhesion molecule alpha catenin beta catenin BMI1 protein CD133 antigen CD24 antigen cell surface marker endoglin fibronectin glycogen synthase kinase 3beta Hermes antigen membrane antigen messenger RNA Notch1 receptor octamer transcription factor 4 protein kinase B short hairpin RNA transcription factor NANOG transcription factor Snail uvomorulin vimentin adult animal experiment animal model animal tissue article cancer recurrence cancer resistance cancer stem cell cancer survival carcinogenesis carcinogenicity cell growth cell migration cell motility cell renewal chemosensitivity chromosome 1q controlled study enzyme activity epithelial mesenchymal transition female gene amplification gene overexpression gene silencing human human cell human tissue liver cell carcinoma Maelstrom gene male metastasis middle aged mouse multidrug resistance nonhuman oncogene overall survival priority journal RNA interference signal transduction tumor invasion tumor volume tumor xenograft upregulation Animals Carcinoma, Hepatocellular Carrier Proteins Cell Movement Cell Proliferation Disease Models, Animal Disease Progression Epithelial-Mesenchymal Transition Female Glycogen Synthase Kinase 3 Humans Liver Neoplasms Male Mice Middle Aged Neoplasm Metastasis Proto-Oncogene Proteins c-akt RNA, Messenger Signal Transduction Transcription Factors Up-Regulation 10.1002/hep.26677 Hepatology 59 2 531-543 2020-10-27T04:41:23Z 2020-10-27T04:41:23Z 2014 Article Liu, L, Dai, Y, Chen, J, Zeng, T, Li, Y, Chen, L, Zhu, Y.-H, Li, J, Li, Y, Ma, S, Xie, D, Yuan, Y.-F, Guan, X.-Y (2014). Maelstrom promotes hepatocellular carcinoma metastasis by inducing epithelial-mesenchymal transition by way of Akt/GSK-3β/Snail signaling. Hepatology 59 (2) : 531-543. ScholarBank@NUS Repository. https://doi.org/10.1002/hep.26677 0270-9139 https://scholarbank.nus.edu.sg/handle/10635/180768 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ John Wiley and Sons Inc. Unpaywall 20201031
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	activated leukocyte cell adhesion molecule alpha catenin beta catenin BMI1 protein CD133 antigen CD24 antigen cell surface marker endoglin fibronectin glycogen synthase kinase 3beta Hermes antigen membrane antigen messenger RNA Notch1 receptor octamer transcription factor 4 protein kinase B short hairpin RNA transcription factor NANOG transcription factor Snail uvomorulin vimentin adult animal experiment animal model animal tissue article cancer recurrence cancer resistance cancer stem cell cancer survival carcinogenesis carcinogenicity cell growth cell migration cell motility cell renewal chemosensitivity chromosome 1q controlled study enzyme activity epithelial mesenchymal transition female gene amplification gene overexpression gene silencing human human cell human tissue liver cell carcinoma Maelstrom gene male metastasis middle aged mouse multidrug resistance nonhuman oncogene overall survival priority journal RNA interference signal transduction tumor invasion tumor volume tumor xenograft upregulation Animals Carcinoma, Hepatocellular Carrier Proteins Cell Movement Cell Proliferation Disease Models, Animal Disease Progression Epithelial-Mesenchymal Transition Female Glycogen Synthase Kinase 3 Humans Liver Neoplasms Male Mice Middle Aged Neoplasm Metastasis Proto-Oncogene Proteins c-akt RNA, Messenger Signal Transduction Transcription Factors Up-Regulation
spellingShingle	activated leukocyte cell adhesion molecule alpha catenin beta catenin BMI1 protein CD133 antigen CD24 antigen cell surface marker endoglin fibronectin glycogen synthase kinase 3beta Hermes antigen membrane antigen messenger RNA Notch1 receptor octamer transcription factor 4 protein kinase B short hairpin RNA transcription factor NANOG transcription factor Snail uvomorulin vimentin adult animal experiment animal model animal tissue article cancer recurrence cancer resistance cancer stem cell cancer survival carcinogenesis carcinogenicity cell growth cell migration cell motility cell renewal chemosensitivity chromosome 1q controlled study enzyme activity epithelial mesenchymal transition female gene amplification gene overexpression gene silencing human human cell human tissue liver cell carcinoma Maelstrom gene male metastasis middle aged mouse multidrug resistance nonhuman oncogene overall survival priority journal RNA interference signal transduction tumor invasion tumor volume tumor xenograft upregulation Animals Carcinoma, Hepatocellular Carrier Proteins Cell Movement Cell Proliferation Disease Models, Animal Disease Progression Epithelial-Mesenchymal Transition Female Glycogen Synthase Kinase 3 Humans Liver Neoplasms Male Mice Middle Aged Neoplasm Metastasis Proto-Oncogene Proteins c-akt RNA, Messenger Signal Transduction Transcription Factors Up-Regulation Liu, L Dai, Y Chen, J Zeng, T Li, Y Chen, L Zhu, Y.-H Li, J Li, Y Ma, S Xie, D Yuan, Y.-F Guan, X.-Y Maelstrom promotes hepatocellular carcinoma metastasis by inducing epithelial-mesenchymal transition by way of Akt/GSK-3β/Snail signaling
description	10.1002/hep.26677
author2	ANATOMY
author_facet	ANATOMY Liu, L Dai, Y Chen, J Zeng, T Li, Y Chen, L Zhu, Y.-H Li, J Li, Y Ma, S Xie, D Yuan, Y.-F Guan, X.-Y
format	Article
author	Liu, L Dai, Y Chen, J Zeng, T Li, Y Chen, L Zhu, Y.-H Li, J Li, Y Ma, S Xie, D Yuan, Y.-F Guan, X.-Y
author_sort	Liu, L
title	Maelstrom promotes hepatocellular carcinoma metastasis by inducing epithelial-mesenchymal transition by way of Akt/GSK-3β/Snail signaling
title_short	Maelstrom promotes hepatocellular carcinoma metastasis by inducing epithelial-mesenchymal transition by way of Akt/GSK-3β/Snail signaling
title_full	Maelstrom promotes hepatocellular carcinoma metastasis by inducing epithelial-mesenchymal transition by way of Akt/GSK-3β/Snail signaling
title_fullStr	Maelstrom promotes hepatocellular carcinoma metastasis by inducing epithelial-mesenchymal transition by way of Akt/GSK-3β/Snail signaling
title_full_unstemmed	Maelstrom promotes hepatocellular carcinoma metastasis by inducing epithelial-mesenchymal transition by way of Akt/GSK-3β/Snail signaling
title_sort	maelstrom promotes hepatocellular carcinoma metastasis by inducing epithelial-mesenchymal transition by way of akt/gsk-3β/snail signaling
publisher	John Wiley and Sons Inc.
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/180768
_version_	1821193595870248960

Maelstrom promotes hepatocellular carcinoma metastasis by inducing epithelial-mesenchymal transition by way of Akt/GSK-3β/Snail signaling

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