HPV16-E2 induces prophase arrest and activates the cellular DNA damage response in vitro and in precursor lesions of cervical carcinoma

10.18632/oncotarget.5512

Saved in:

Bibliographic Details
Main Authors:	Xue, Y, Toh, S.Y, He, P, Lim, T, Lim, D, Pang, C.L, Abastado, J.-P, Thierry, F
Other Authors:	PATHOLOGY
Format:	Article
Published:	2020
Subjects:	DNA unclassified drug virus E2 protein virus protein DNA binding protein E2 protein, Human papillomavirus type 16 oncoprotein Article cell cycle arrest cell cycle regulation controlled study DNA damage DNA repair female genomic instability human human cell Human papillomavirus type 16 human tissue in vitro study in vivo study major clinical study malignant transformation nonhuman prophase protein expression uterine cervix carcinoma in situ virus carcinogenesis virus cell interaction cell cycle checkpoint cell transformation complication flow cytometry fluorescent antibody technique immunohistochemistry immunoprecipitation in situ hybridization metabolism papillomavirus infection pathology physiology precancer uterine cervix tumor virology Western blotting Blotting, Western Cell Cycle Checkpoints Cell Transformation, Neoplastic Cervical Intraepithelial Neoplasia DNA Damage DNA-Binding Proteins Female Flow Cytometry Fluorescent Antibody Technique Humans Immunohistochemistry Immunoprecipitation In Situ Hybridization Oncogene Proteins, Viral Papillomavirus Infections Precancerous Conditions Prophase Uterine Cervical Neoplasms
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/180931
Tags:	Add Tag No Tags, Be the first to tag this record!
Institution:	National University of Singapore

id	sg-nus-scholar.10635-180931
record_format	dspace
spelling	sg-nus-scholar.10635-1809312024-04-03T07:38:19Z HPV16-E2 induces prophase arrest and activates the cellular DNA damage response in vitro and in precursor lesions of cervical carcinoma Xue, Y Toh, S.Y He, P Lim, T Lim, D Pang, C.L Abastado, J.-P Thierry, F PATHOLOGY DNA unclassified drug virus E2 protein virus protein DNA binding protein E2 protein, Human papillomavirus type 16 oncoprotein Article cell cycle arrest cell cycle regulation controlled study DNA damage DNA repair female genomic instability human human cell Human papillomavirus type 16 human tissue in vitro study in vivo study major clinical study malignant transformation nonhuman prophase protein expression uterine cervix carcinoma in situ virus carcinogenesis virus cell interaction cell cycle checkpoint cell transformation complication flow cytometry fluorescent antibody technique immunohistochemistry immunoprecipitation in situ hybridization metabolism papillomavirus infection pathology physiology precancer prophase uterine cervix carcinoma in situ uterine cervix tumor virology Western blotting Blotting, Western Cell Cycle Checkpoints Cell Transformation, Neoplastic Cervical Intraepithelial Neoplasia DNA Damage DNA-Binding Proteins Female Flow Cytometry Fluorescent Antibody Technique Humans Immunohistochemistry Immunoprecipitation In Situ Hybridization Oncogene Proteins, Viral Papillomavirus Infections Precancerous Conditions Prophase Uterine Cervical Neoplasms 10.18632/oncotarget.5512 Oncotarget 6 33 34979-34991 2020-10-27T05:45:16Z 2020-10-27T05:45:16Z 2015 Article Xue, Y, Toh, S.Y, He, P, Lim, T, Lim, D, Pang, C.L, Abastado, J.-P, Thierry, F (2015). HPV16-E2 induces prophase arrest and activates the cellular DNA damage response in vitro and in precursor lesions of cervical carcinoma. Oncotarget 6 (33) : 34979-34991. ScholarBank@NUS Repository. https://doi.org/10.18632/oncotarget.5512 19492553 https://scholarbank.nus.edu.sg/handle/10635/180931 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ Unpaywall 20201031
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	DNA unclassified drug virus E2 protein virus protein DNA binding protein E2 protein, Human papillomavirus type 16 oncoprotein Article cell cycle arrest cell cycle regulation controlled study DNA damage DNA repair female genomic instability human human cell Human papillomavirus type 16 human tissue in vitro study in vivo study major clinical study malignant transformation nonhuman prophase protein expression uterine cervix carcinoma in situ virus carcinogenesis virus cell interaction cell cycle checkpoint cell transformation complication flow cytometry fluorescent antibody technique immunohistochemistry immunoprecipitation in situ hybridization metabolism papillomavirus infection pathology physiology precancer prophase uterine cervix carcinoma in situ uterine cervix tumor virology Western blotting Blotting, Western Cell Cycle Checkpoints Cell Transformation, Neoplastic Cervical Intraepithelial Neoplasia DNA Damage DNA-Binding Proteins Female Flow Cytometry Fluorescent Antibody Technique Humans Immunohistochemistry Immunoprecipitation In Situ Hybridization Oncogene Proteins, Viral Papillomavirus Infections Precancerous Conditions Prophase Uterine Cervical Neoplasms
spellingShingle	DNA unclassified drug virus E2 protein virus protein DNA binding protein E2 protein, Human papillomavirus type 16 oncoprotein Article cell cycle arrest cell cycle regulation controlled study DNA damage DNA repair female genomic instability human human cell Human papillomavirus type 16 human tissue in vitro study in vivo study major clinical study malignant transformation nonhuman prophase protein expression uterine cervix carcinoma in situ virus carcinogenesis virus cell interaction cell cycle checkpoint cell transformation complication flow cytometry fluorescent antibody technique immunohistochemistry immunoprecipitation in situ hybridization metabolism papillomavirus infection pathology physiology precancer prophase uterine cervix carcinoma in situ uterine cervix tumor virology Western blotting Blotting, Western Cell Cycle Checkpoints Cell Transformation, Neoplastic Cervical Intraepithelial Neoplasia DNA Damage DNA-Binding Proteins Female Flow Cytometry Fluorescent Antibody Technique Humans Immunohistochemistry Immunoprecipitation In Situ Hybridization Oncogene Proteins, Viral Papillomavirus Infections Precancerous Conditions Prophase Uterine Cervical Neoplasms Xue, Y Toh, S.Y He, P Lim, T Lim, D Pang, C.L Abastado, J.-P Thierry, F HPV16-E2 induces prophase arrest and activates the cellular DNA damage response in vitro and in precursor lesions of cervical carcinoma
description	10.18632/oncotarget.5512
author2	PATHOLOGY
author_facet	PATHOLOGY Xue, Y Toh, S.Y He, P Lim, T Lim, D Pang, C.L Abastado, J.-P Thierry, F
format	Article
author	Xue, Y Toh, S.Y He, P Lim, T Lim, D Pang, C.L Abastado, J.-P Thierry, F
author_sort	Xue, Y
title	HPV16-E2 induces prophase arrest and activates the cellular DNA damage response in vitro and in precursor lesions of cervical carcinoma
title_short	HPV16-E2 induces prophase arrest and activates the cellular DNA damage response in vitro and in precursor lesions of cervical carcinoma
title_full	HPV16-E2 induces prophase arrest and activates the cellular DNA damage response in vitro and in precursor lesions of cervical carcinoma
title_fullStr	HPV16-E2 induces prophase arrest and activates the cellular DNA damage response in vitro and in precursor lesions of cervical carcinoma
title_full_unstemmed	HPV16-E2 induces prophase arrest and activates the cellular DNA damage response in vitro and in precursor lesions of cervical carcinoma
title_sort	hpv16-e2 induces prophase arrest and activates the cellular dna damage response in vitro and in precursor lesions of cervical carcinoma
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/180931
_version_	1795374527383863296

HPV16-E2 induces prophase arrest and activates the cellular DNA damage response in vitro and in precursor lesions of cervical carcinoma

Similar Items