SIRT1 in B[a]P-induced lung tumorigenesis

• المؤلفون الرئيسيون: Lu, J, Zhang, M, Huang, Z, Sun, S, Zhang, Y, Zhang, L, Peng, L, Ma, A, Ji, P, Dai, J, Cui, T, Liu, H, Gao, J
• مؤلفون آخرون: DEPARTMENT OF COMPUTER SCIENCE
• التنسيق: مقال
• منشور في: 2020
• الموضوعات: beta catenin; sirtuin 1; tumor necrosis factor alpha; uvomorulin; benzo[a]pyrene; SIRT1 protein, human; tumor necrosis factor; animal experiment; animal model; animal tissue; Article; bronchus mucosa; cell invasion; cell migration; controlled study; down regulation; epithelium cell; human; human cell; human tissue; lung carcinogenesis; mouse; nonhuman; protein expression; protein function; protein protein interaction; protein targeting; tumor biopsy; upregulation; Wnt signaling pathway; adverse effects; animal; biopsy; bronchus; C57BL mouse; carcinogenesis; cell motion; cell transformation; chemically induced; chemistry; female; genetic transcription; lung; lung tumor; metabolism; nude mouse; pathology; smoking; tumor cell line; tumor invasion; Animals; Benzo(a)pyrene; beta Catenin; Biopsy; Bronchi; Carcinogenesis; Cell Line, Tumor; Cell Movement; Cell Transformation, Neoplastic; Epithelial Cells; Female; Humans; Lung; Lung Neoplasms; Mice; Mice, Inbred C57BL; Mice, Nude; Neoplasm Invasiveness; Sirtuin 1; Smoking; Transcription, Genetic; Tumor Necrosis Factor-alpha; Up-Regulation
• الوصول للمادة أونلاين: https://scholarbank.nus.edu.sg/handle/10635/180937

10.18632/oncotarget.4729