The transcription factor CREM drives an inflammatory phenotype of T cells in oligoarticular juvenile idiopathic arthritis

10.1186/s12969-018-0253-x

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Bibliographic Details
Main Authors:	Ohl, K, Nickel, H, Moncrieffe, H, Klemm, P, Scheufen, A, Föll, D, Wixler, V, Schippers, A, Wagner, N, Wedderburn, L.R, Tenbrock, K
Other Authors:	SAW SWEE HOCK SCHOOL OF PUBLIC HEALTH
Format:	Article
Published:	2020
Subjects:	beta3 integrin cyclic AMP responsive element modulator gamma interferon interleukin 17 ovalbumin small interfering RNA transcription factor FOXP3 CREM protein, human adoptive transfer animal tissue Article cellular immunity controlled study flow cytometry human human cell immunopathogenesis in vivo study incubation time inflammation juvenile rheumatoid arthritis mouse nonhuman oligoarticular juvenile idiopathic arthritis peripheral blood mononuclear cell phenotype priority journal protein expression protein function regulatory T lymphocyte synovial fluid T lymphocyte subpopulation upregulation animal cell culture technique joint metabolism pathology procedures real time polymerase chain reaction T lymphocyte Animals Arthritis, Juvenile Cell Culture Techniques Cyclic AMP Response Element Modulator Flow Cytometry Humans Joints Mice Phenotype Real-Time Polymerase Chain Reaction Synovial Fluid T-Lymphocytes
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/181196
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Institution:	National University of Singapore

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