Trefoil factor 3 promotes metastatic seeding and predicts poor survival outcome of patients with mammary carcinoma

• المؤلفون الرئيسيون: Pandey, V, Wu, Z.-S, Zhang, M, Li, R, Zhang, J, Zhu, T, Lobie, P.E
• مؤلفون آخرون: PHARMACOLOGY
• التنسيق: مقال
• منشور في: 2020
• الموضوعات: estrogen receptor; messenger RNA; protein tyrosine kinase; STAT3 protein; trefoil factor 3 protein; trefoil peptide; unclassified drug; uvomorulin; cadherin; CDH1 protein, human; CSK tyrosine-protein kinase; peptide; STAT3 protein, human; TFF3 protein, human; adult; animal experiment; animal model; animal tissue; Article; breast cancer cell line; breast carcinoma; cancer prognosis; cancer staging; cell adhesion; cell invasion; cell migration; colony formation; controlled study; down regulation; endothelium cell; gene expression; human; human cell; human tissue; lung metastasis; lymph node metastasis; major clinical study; marker gene; metastasis; mouse; nonhuman; oncogene; overall survival; protein expression; protein phosphorylation; recurrence free survival; tumor volume; animal; Bagg albino mouse; breast tumor; cancer transplantation; disease free survival; female; genetics; Kaplan Meier method; lung tumor; MCF 7 cell line; metabolism; middle aged; mortality; nude mouse; Paget nipple disease; pathology; phosphorylation; physiology; proportional hazards model; protein processing; secondary; tumor invasion; Animals; Breast Neoplasms; Cadherins; Carcinoma, Ductal, Breast; Disease-Free Survival; Female; Gene Expression; Humans; Kaplan-Meier Estimate; Lung Neoplasms; Lymphatic Metastasis; MCF-7 Cells; Mice, Inbred BALB C; Mice, Nude; Middle Aged; Neoplasm Invasiveness; Neoplasm Transplantation; Peptides; Phosphorylation; Proportional Hazards Models; Protein Processing, Post-Translational; src-Family Kinases; STAT3 Transcription Factor
• الوصول للمادة أونلاين: https://scholarbank.nus.edu.sg/handle/10635/183688

10.1186/s13058-014-0429-3