Decreased GLUT2 and glucose uptake contribute to insulin secretion defects in MODY3/HNF1A hiPSC-derived mutant ? cells

10.1038/s41467-021-22843-4

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Main Authors: Low, Blaise Su Jun, Lim, Chang Siang, Ding, Shirley Suet Lee, Tan, Yaw Sing, Ng, Natasha Hui Jin, Krishnan, Vidhya Gomathi, Ang, Su Fen, Neo, Claire Wen Ying, Verma, Chandra S., Hoon, Shawn, Lim, Su Chi, Tai, E. Shyong, Teo, Adrian Kee Keong
Other Authors: DEAN'S OFFICE (SSH SCH OF PUBLIC HEALTH)
Format: Article
Published: Nature Research 2022
Online Access:https://scholarbank.nus.edu.sg/handle/10635/232740
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spelling sg-nus-scholar.10635-2327402024-04-18T02:01:08Z Decreased GLUT2 and glucose uptake contribute to insulin secretion defects in MODY3/HNF1A hiPSC-derived mutant ? cells Low, Blaise Su Jun Lim, Chang Siang Ding, Shirley Suet Lee Tan, Yaw Sing Ng, Natasha Hui Jin Krishnan, Vidhya Gomathi Ang, Su Fen Neo, Claire Wen Ying Verma, Chandra S. Hoon, Shawn Lim, Su Chi Tai, E. Shyong Teo, Adrian Kee Keong DEAN'S OFFICE (SSH SCH OF PUBLIC HEALTH) BIOLOGICAL SCIENCES SAW SWEE HOCK SCHOOL OF PUBLIC HEALTH MEDICINE BIOCHEMISTRY 10.1038/s41467-021-22843-4 Nature Communications 12 1 3133 2022-10-13T01:07:36Z 2022-10-13T01:07:36Z 2021-05-25 Article Low, Blaise Su Jun, Lim, Chang Siang, Ding, Shirley Suet Lee, Tan, Yaw Sing, Ng, Natasha Hui Jin, Krishnan, Vidhya Gomathi, Ang, Su Fen, Neo, Claire Wen Ying, Verma, Chandra S., Hoon, Shawn, Lim, Su Chi, Tai, E. Shyong, Teo, Adrian Kee Keong (2021-05-25). Decreased GLUT2 and glucose uptake contribute to insulin secretion defects in MODY3/HNF1A hiPSC-derived mutant ? cells. Nature Communications 12 (1) : 3133. ScholarBank@NUS Repository. https://doi.org/10.1038/s41467-021-22843-4 2041-1723 https://scholarbank.nus.edu.sg/handle/10635/232740 Attribution 4.0 International https://creativecommons.org/licenses/by/4.0/ Nature Research Scopus OA2021
institution National University of Singapore
building NUS Library
continent Asia
country Singapore
Singapore
content_provider NUS Library
collection ScholarBank@NUS
description 10.1038/s41467-021-22843-4
author2 DEAN'S OFFICE (SSH SCH OF PUBLIC HEALTH)
author_facet DEAN'S OFFICE (SSH SCH OF PUBLIC HEALTH)
Low, Blaise Su Jun
Lim, Chang Siang
Ding, Shirley Suet Lee
Tan, Yaw Sing
Ng, Natasha Hui Jin
Krishnan, Vidhya Gomathi
Ang, Su Fen
Neo, Claire Wen Ying
Verma, Chandra S.
Hoon, Shawn
Lim, Su Chi
Tai, E. Shyong
Teo, Adrian Kee Keong
format Article
author Low, Blaise Su Jun
Lim, Chang Siang
Ding, Shirley Suet Lee
Tan, Yaw Sing
Ng, Natasha Hui Jin
Krishnan, Vidhya Gomathi
Ang, Su Fen
Neo, Claire Wen Ying
Verma, Chandra S.
Hoon, Shawn
Lim, Su Chi
Tai, E. Shyong
Teo, Adrian Kee Keong
spellingShingle Low, Blaise Su Jun
Lim, Chang Siang
Ding, Shirley Suet Lee
Tan, Yaw Sing
Ng, Natasha Hui Jin
Krishnan, Vidhya Gomathi
Ang, Su Fen
Neo, Claire Wen Ying
Verma, Chandra S.
Hoon, Shawn
Lim, Su Chi
Tai, E. Shyong
Teo, Adrian Kee Keong
Decreased GLUT2 and glucose uptake contribute to insulin secretion defects in MODY3/HNF1A hiPSC-derived mutant ? cells
author_sort Low, Blaise Su Jun
title Decreased GLUT2 and glucose uptake contribute to insulin secretion defects in MODY3/HNF1A hiPSC-derived mutant ? cells
title_short Decreased GLUT2 and glucose uptake contribute to insulin secretion defects in MODY3/HNF1A hiPSC-derived mutant ? cells
title_full Decreased GLUT2 and glucose uptake contribute to insulin secretion defects in MODY3/HNF1A hiPSC-derived mutant ? cells
title_fullStr Decreased GLUT2 and glucose uptake contribute to insulin secretion defects in MODY3/HNF1A hiPSC-derived mutant ? cells
title_full_unstemmed Decreased GLUT2 and glucose uptake contribute to insulin secretion defects in MODY3/HNF1A hiPSC-derived mutant ? cells
title_sort decreased glut2 and glucose uptake contribute to insulin secretion defects in mody3/hnf1a hipsc-derived mutant ? cells
publisher Nature Research
publishDate 2022
url https://scholarbank.nus.edu.sg/handle/10635/232740
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