SETDB1 acts as a topological accessory to Cohesin via an H3K9me3-independent, genomic shunt for regulating cell fates

SETDB1 acts as a topological accessory to Cohesin via an H3K9me3-independent, genomic shunt for regulating cell fates

10.1093/nar/gkac531

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Bibliographic Details
Main Authors:	Warrier, T, El Farran, C, Zeng, Y, Ho, BSQ, Bao, Q, Zheng, ZH, Bi, X, Ng, HH, Ong, DST, Chu, JJH, Sanyal, A, Fullwood, MJ, Collins, JJ, Li, H, Xu, J, Loh, YH
Other Authors:	BIOLOGY (NU)
Format:	Article
Published:	Oxford University Press (OUP) 2022
Subjects:	Animals Cell Cycle Proteins Chromosomal Proteins, Non-Histone Endogenous Retroviruses Genomics Histone-Lysine N-Methyltransferase Histones Mice
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/235419
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Institution:	National University of Singapore

Description
Summary:	10.1093/nar/gkac531