SETDB1 acts as a topological accessory to Cohesin via an H3K9me3-independent, genomic shunt for regulating cell fates

10.1093/nar/gkac531

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Main Authors: Warrier, T, El Farran, C, Zeng, Y, Ho, BSQ, Bao, Q, Zheng, ZH, Bi, X, Ng, HH, Ong, DST, Chu, JJH, Sanyal, A, Fullwood, MJ, Collins, JJ, Li, H, Xu, J, Loh, YH
Other Authors: BIOLOGY (NU)
Format: Article
Published: Oxford University Press (OUP) 2022
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Online Access:https://scholarbank.nus.edu.sg/handle/10635/235419
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spelling sg-nus-scholar.10635-2354192024-04-04T00:53:27Z SETDB1 acts as a topological accessory to Cohesin via an H3K9me3-independent, genomic shunt for regulating cell fates Warrier, T El Farran, C Zeng, Y Ho, BSQ Bao, Q Zheng, ZH Bi, X Ng, HH Ong, DST Chu, JJH Sanyal, A Fullwood, MJ Collins, JJ Li, H Xu, J Loh, YH BIOLOGY (NU) CANCER SCIENCE INSTITUTE OF SINGAPORE BIOLOGICAL SCIENCES MICROBIOLOGY AND IMMUNOLOGY PHYSIOLOGY Animals Cell Cycle Proteins Chromosomal Proteins, Non-Histone Endogenous Retroviruses Genomics Histone-Lysine N-Methyltransferase Histones Mice 10.1093/nar/gkac531 Nucleic Acids Research 50 13 7326-7349 2022-12-08T07:24:39Z 2022-12-08T07:24:39Z 2022-07-22 2022-12-08T05:26:21Z Article Warrier, T, El Farran, C, Zeng, Y, Ho, BSQ, Bao, Q, Zheng, ZH, Bi, X, Ng, HH, Ong, DST, Chu, JJH, Sanyal, A, Fullwood, MJ, Collins, JJ, Li, H, Xu, J, Loh, YH (2022-07-22). SETDB1 acts as a topological accessory to Cohesin via an H3K9me3-independent, genomic shunt for regulating cell fates. Nucleic Acids Research 50 (13) : 7326-7349. ScholarBank@NUS Repository. https://doi.org/10.1093/nar/gkac531 0305-1048 1362-4962 https://scholarbank.nus.edu.sg/handle/10635/235419 Oxford University Press (OUP) Elements
institution National University of Singapore
building NUS Library
continent Asia
country Singapore
Singapore
content_provider NUS Library
collection ScholarBank@NUS
topic Animals
Cell Cycle Proteins
Chromosomal Proteins, Non-Histone
Endogenous Retroviruses
Genomics
Histone-Lysine N-Methyltransferase
Histones
Mice
spellingShingle Animals
Cell Cycle Proteins
Chromosomal Proteins, Non-Histone
Endogenous Retroviruses
Genomics
Histone-Lysine N-Methyltransferase
Histones
Mice
Warrier, T
El Farran, C
Zeng, Y
Ho, BSQ
Bao, Q
Zheng, ZH
Bi, X
Ng, HH
Ong, DST
Chu, JJH
Sanyal, A
Fullwood, MJ
Collins, JJ
Li, H
Xu, J
Loh, YH
SETDB1 acts as a topological accessory to Cohesin via an H3K9me3-independent, genomic shunt for regulating cell fates
description 10.1093/nar/gkac531
author2 BIOLOGY (NU)
author_facet BIOLOGY (NU)
Warrier, T
El Farran, C
Zeng, Y
Ho, BSQ
Bao, Q
Zheng, ZH
Bi, X
Ng, HH
Ong, DST
Chu, JJH
Sanyal, A
Fullwood, MJ
Collins, JJ
Li, H
Xu, J
Loh, YH
format Article
author Warrier, T
El Farran, C
Zeng, Y
Ho, BSQ
Bao, Q
Zheng, ZH
Bi, X
Ng, HH
Ong, DST
Chu, JJH
Sanyal, A
Fullwood, MJ
Collins, JJ
Li, H
Xu, J
Loh, YH
author_sort Warrier, T
title SETDB1 acts as a topological accessory to Cohesin via an H3K9me3-independent, genomic shunt for regulating cell fates
title_short SETDB1 acts as a topological accessory to Cohesin via an H3K9me3-independent, genomic shunt for regulating cell fates
title_full SETDB1 acts as a topological accessory to Cohesin via an H3K9me3-independent, genomic shunt for regulating cell fates
title_fullStr SETDB1 acts as a topological accessory to Cohesin via an H3K9me3-independent, genomic shunt for regulating cell fates
title_full_unstemmed SETDB1 acts as a topological accessory to Cohesin via an H3K9me3-independent, genomic shunt for regulating cell fates
title_sort setdb1 acts as a topological accessory to cohesin via an h3k9me3-independent, genomic shunt for regulating cell fates
publisher Oxford University Press (OUP)
publishDate 2022
url https://scholarbank.nus.edu.sg/handle/10635/235419
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