Enhanced activation of STAT pathways and overexpression of survivin confer resistance to FLT3 inhibitors and could be therapeutic targets in AML
Blood
Saved in:
Main Authors: | Zhou, J., Bi, C., Janakakumara, J.V., Liu, S.-C., Chng, W.-J., Tay, K.-G., Poon, L.-F., Xie, Z., Palaniyandi, S., Chen, C.-S., Yu, H., Glaser, K.B., Albert, D.H., Davidsen, S.K. |
---|---|
Other Authors: | PHYSIOLOGY |
Format: | Article |
Published: |
2011
|
Online Access: | http://scholarbank.nus.edu.sg/handle/10635/25105 |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Institution: | National University of Singapore |
Similar Items
-
Enhanced activation of STAT pathways and overexpression of survivin confer resistance to FLT3 inhibitors and could be therapeutic targets in AML
by: Zhou, J., et al.
Published: (2011) -
Overexpression of survivin and caspase 3 in oral carcinogenesis
by: Sopee Poomsawat, et al.
Published: (2018) -
PRL-3, a metastasis associated tyrosine phosphatase, is involved in FLT3-ITD signaling and implicated in anti-aml therapy
by: Zhou, J., et al.
Published: (2014) -
In vivo activity of ABT-869, a multi-target kinase inhibitor, against acute myeloid leukemia with wild-type FLT3 receptor
by: Zhou, J., et al.
Published: (2011) -
In vivo activity of ABT-869, a multi-target kinase inhibitor, against acute myeloid leukemia with wild-type FLT3 receptor
by: Zhou, J., et al.
Published: (2011)