The highly pathogenic H7N3 avian influenza strain from July 2012 in Mexico acquired an extended cleavage site through recombination with host 28S rRNA

• Main Authors: Maurer-Stroh, S, Lee, R.T, Gunalan, V, Eisenhaber, F
• Other Authors: BIOLOGICAL SCIENCES
• Format: Article
• Published: 2020
• Subjects: RNA 28S; article; gene sequence; host; Influenza virus A H7N3; Mexico; nonhuman; pathogenesis; pathogenicity; phylogeny; poultry; RNA analysis; RNA cleavage; RNA sequence; RNA structure; virus strain; virus transmission; Animals; Chickens; Hemagglutinin Glycoproteins, Influenza Virus; Influenza A Virus, H7N3 Subtype; Influenza in Birds; Phylogeny; Recombination, Genetic; RNA, Ribosomal, 28S; RNA, Viral; Sequence Analysis, DNA; Aves; Mammalia
• Online Access: https://scholarbank.nus.edu.sg/handle/10635/174175
• Institution: National University of Singapore

10.1186/1743-422X-10-139