The highly pathogenic H7N3 avian influenza strain from July 2012 in Mexico acquired an extended cleavage site through recombination with host 28S rRNA

10.1186/1743-422X-10-139

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Main Authors: Maurer-Stroh, S, Lee, R.T, Gunalan, V, Eisenhaber, F
Other Authors: BIOLOGICAL SCIENCES
Format: Article
Published: 2020
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Online Access:https://scholarbank.nus.edu.sg/handle/10635/174175
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spelling sg-nus-scholar.10635-1741752024-04-03T06:05:25Z The highly pathogenic H7N3 avian influenza strain from July 2012 in Mexico acquired an extended cleavage site through recombination with host 28S rRNA Maurer-Stroh, S Lee, R.T Gunalan, V Eisenhaber, F BIOLOGICAL SCIENCES RNA 28S article gene sequence host Influenza virus A H7N3 Mexico nonhuman pathogenesis pathogenicity phylogeny poultry RNA analysis RNA cleavage RNA sequence RNA structure virus strain virus transmission Animals Chickens Hemagglutinin Glycoproteins, Influenza Virus Influenza A Virus, H7N3 Subtype Influenza in Birds Mexico Phylogeny Recombination, Genetic RNA, Ribosomal, 28S RNA, Viral Sequence Analysis, DNA Aves Mammalia 10.1186/1743-422X-10-139 Virology Journal 10 139 2020-09-03T10:43:32Z 2020-09-03T10:43:32Z 2013 Article Maurer-Stroh, S, Lee, R.T, Gunalan, V, Eisenhaber, F (2013). The highly pathogenic H7N3 avian influenza strain from July 2012 in Mexico acquired an extended cleavage site through recombination with host 28S rRNA. Virology Journal 10 : 139. ScholarBank@NUS Repository. https://doi.org/10.1186/1743-422X-10-139 1743422X https://scholarbank.nus.edu.sg/handle/10635/174175 Unpaywall 20200831
institution National University of Singapore
building NUS Library
continent Asia
country Singapore
Singapore
content_provider NUS Library
collection ScholarBank@NUS
topic RNA 28S
article
gene sequence
host
Influenza virus A H7N3
Mexico
nonhuman
pathogenesis
pathogenicity
phylogeny
poultry
RNA analysis
RNA cleavage
RNA sequence
RNA structure
virus strain
virus transmission
Animals
Chickens
Hemagglutinin Glycoproteins, Influenza Virus
Influenza A Virus, H7N3 Subtype
Influenza in Birds
Mexico
Phylogeny
Recombination, Genetic
RNA, Ribosomal, 28S
RNA, Viral
Sequence Analysis, DNA
Aves
Mammalia
spellingShingle RNA 28S
article
gene sequence
host
Influenza virus A H7N3
Mexico
nonhuman
pathogenesis
pathogenicity
phylogeny
poultry
RNA analysis
RNA cleavage
RNA sequence
RNA structure
virus strain
virus transmission
Animals
Chickens
Hemagglutinin Glycoproteins, Influenza Virus
Influenza A Virus, H7N3 Subtype
Influenza in Birds
Mexico
Phylogeny
Recombination, Genetic
RNA, Ribosomal, 28S
RNA, Viral
Sequence Analysis, DNA
Aves
Mammalia
Maurer-Stroh, S
Lee, R.T
Gunalan, V
Eisenhaber, F
The highly pathogenic H7N3 avian influenza strain from July 2012 in Mexico acquired an extended cleavage site through recombination with host 28S rRNA
description 10.1186/1743-422X-10-139
author2 BIOLOGICAL SCIENCES
author_facet BIOLOGICAL SCIENCES
Maurer-Stroh, S
Lee, R.T
Gunalan, V
Eisenhaber, F
format Article
author Maurer-Stroh, S
Lee, R.T
Gunalan, V
Eisenhaber, F
author_sort Maurer-Stroh, S
title The highly pathogenic H7N3 avian influenza strain from July 2012 in Mexico acquired an extended cleavage site through recombination with host 28S rRNA
title_short The highly pathogenic H7N3 avian influenza strain from July 2012 in Mexico acquired an extended cleavage site through recombination with host 28S rRNA
title_full The highly pathogenic H7N3 avian influenza strain from July 2012 in Mexico acquired an extended cleavage site through recombination with host 28S rRNA
title_fullStr The highly pathogenic H7N3 avian influenza strain from July 2012 in Mexico acquired an extended cleavage site through recombination with host 28S rRNA
title_full_unstemmed The highly pathogenic H7N3 avian influenza strain from July 2012 in Mexico acquired an extended cleavage site through recombination with host 28S rRNA
title_sort highly pathogenic h7n3 avian influenza strain from july 2012 in mexico acquired an extended cleavage site through recombination with host 28s rrna
publishDate 2020
url https://scholarbank.nus.edu.sg/handle/10635/174175
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