The highly pathogenic H7N3 avian influenza strain from July 2012 in Mexico acquired an extended cleavage site through recombination with host 28S rRNA

The highly pathogenic H7N3 avian influenza strain from July 2012 in Mexico acquired an extended cleavage site through recombination with host 28S rRNA

10.1186/1743-422X-10-139

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Bibliographic Details
Main Authors:	Maurer-Stroh, S, Lee, R.T, Gunalan, V, Eisenhaber, F
Other Authors:	BIOLOGICAL SCIENCES
Format:	Article
Published:	2020
Subjects:	RNA 28S article gene sequence host Influenza virus A H7N3 Mexico nonhuman pathogenesis pathogenicity phylogeny poultry RNA analysis RNA cleavage RNA sequence RNA structure virus strain virus transmission Animals Chickens Hemagglutinin Glycoproteins, Influenza Virus Influenza A Virus, H7N3 Subtype Influenza in Birds Phylogeny Recombination, Genetic RNA, Ribosomal, 28S RNA, Viral Sequence Analysis, DNA Aves Mammalia
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/174175
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Institution:	National University of Singapore

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