DNA damage causes rapid accumulation of phosphoinositides for ATR signaling

10.1038/s41467-017-01805-9

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Main Authors:	Wang, Y.-H, Hariharan, A, Bastianello, G, Toyama, Y, Shivashankar, G.V, Foiani, M, Sheetz, M.P
Other Authors:	MECHANOBIOLOGY INSTITUTE
Format:	Article
Published:	Nature Publishing Group 2020
Subjects:	actin ATR protein cell nucleus receptor checkpoint kinase 1 inositol polyphosphate inositol polyphosphate multikinase latrunculin A messenger RNA nuclear protein nuclear receptor protein SF1 phosphatidylinositide unclassified drug wortmannin ATM protein ATR protein, human CHEK1 protein, human phosphatidylinositol phosphotransferase steroidogenic factor 1 chemical reaction DNA enzyme enzyme activity metabolism nervous system disorder protein animal cell Article controlled study DNA damage response DNA repair embryo enzyme activation enzyme phosphorylation gene expression lipid storage mouse nonhuman phosphoinositide metabolism pleckstrin homology domain protein assembly protein binding protein depletion protein expression RNA splicing signal transduction animal cell line DNA damage genetics human RNA interference tumor cell line Animals Ataxia Telangiectasia Mutated Proteins Cell Line Cell Line, Tumor Checkpoint Kinase 1 DNA Damage DNA Repair Humans Mice Phosphatidylinositols Phosphotransferases (Alcohol Group Acceptor) RNA Interference Signal Transduction Steroidogenic Factor 1
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/174372
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Institution:	National University of Singapore

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spelling	sg-nus-scholar.10635-1743722024-04-03T06:27:38Z DNA damage causes rapid accumulation of phosphoinositides for ATR signaling Wang, Y.-H Hariharan, A Bastianello, G Toyama, Y Shivashankar, G.V Foiani, M Sheetz, M.P MECHANOBIOLOGY INSTITUTE BIOLOGICAL SCIENCES BIOLOGY (NU) actin ATR protein cell nucleus receptor checkpoint kinase 1 inositol polyphosphate inositol polyphosphate multikinase latrunculin A messenger RNA nuclear protein nuclear receptor protein SF1 phosphatidylinositide unclassified drug wortmannin ATM protein ATR protein, human checkpoint kinase 1 CHEK1 protein, human inositol polyphosphate multikinase phosphatidylinositol phosphotransferase steroidogenic factor 1 chemical reaction DNA enzyme enzyme activity metabolism nervous system disorder protein animal cell Article controlled study DNA damage response DNA repair embryo enzyme activation enzyme phosphorylation gene expression lipid storage mouse nonhuman phosphoinositide metabolism pleckstrin homology domain protein assembly protein binding protein depletion protein expression RNA splicing signal transduction animal cell line DNA damage genetics human metabolism RNA interference signal transduction tumor cell line Animals Ataxia Telangiectasia Mutated Proteins Cell Line Cell Line, Tumor Checkpoint Kinase 1 DNA Damage DNA Repair Humans Mice Phosphatidylinositols Phosphotransferases (Alcohol Group Acceptor) RNA Interference Signal Transduction Steroidogenic Factor 1 10.1038/s41467-017-01805-9 Nature Communications 8 1 2118 2020-09-04T02:26:35Z 2020-09-04T02:26:35Z 2017 Article Wang, Y.-H, Hariharan, A, Bastianello, G, Toyama, Y, Shivashankar, G.V, Foiani, M, Sheetz, M.P (2017). DNA damage causes rapid accumulation of phosphoinositides for ATR signaling. Nature Communications 8 (1) : 2118. ScholarBank@NUS Repository. https://doi.org/10.1038/s41467-017-01805-9 2041-1723 https://scholarbank.nus.edu.sg/handle/10635/174372 Nature Publishing Group Unpaywall 20200831
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	actin ATR protein cell nucleus receptor checkpoint kinase 1 inositol polyphosphate inositol polyphosphate multikinase latrunculin A messenger RNA nuclear protein nuclear receptor protein SF1 phosphatidylinositide unclassified drug wortmannin ATM protein ATR protein, human checkpoint kinase 1 CHEK1 protein, human inositol polyphosphate multikinase phosphatidylinositol phosphotransferase steroidogenic factor 1 chemical reaction DNA enzyme enzyme activity metabolism nervous system disorder protein animal cell Article controlled study DNA damage response DNA repair embryo enzyme activation enzyme phosphorylation gene expression lipid storage mouse nonhuman phosphoinositide metabolism pleckstrin homology domain protein assembly protein binding protein depletion protein expression RNA splicing signal transduction animal cell line DNA damage genetics human metabolism RNA interference signal transduction tumor cell line Animals Ataxia Telangiectasia Mutated Proteins Cell Line Cell Line, Tumor Checkpoint Kinase 1 DNA Damage DNA Repair Humans Mice Phosphatidylinositols Phosphotransferases (Alcohol Group Acceptor) RNA Interference Signal Transduction Steroidogenic Factor 1
spellingShingle	actin ATR protein cell nucleus receptor checkpoint kinase 1 inositol polyphosphate inositol polyphosphate multikinase latrunculin A messenger RNA nuclear protein nuclear receptor protein SF1 phosphatidylinositide unclassified drug wortmannin ATM protein ATR protein, human checkpoint kinase 1 CHEK1 protein, human inositol polyphosphate multikinase phosphatidylinositol phosphotransferase steroidogenic factor 1 chemical reaction DNA enzyme enzyme activity metabolism nervous system disorder protein animal cell Article controlled study DNA damage response DNA repair embryo enzyme activation enzyme phosphorylation gene expression lipid storage mouse nonhuman phosphoinositide metabolism pleckstrin homology domain protein assembly protein binding protein depletion protein expression RNA splicing signal transduction animal cell line DNA damage genetics human metabolism RNA interference signal transduction tumor cell line Animals Ataxia Telangiectasia Mutated Proteins Cell Line Cell Line, Tumor Checkpoint Kinase 1 DNA Damage DNA Repair Humans Mice Phosphatidylinositols Phosphotransferases (Alcohol Group Acceptor) RNA Interference Signal Transduction Steroidogenic Factor 1 Wang, Y.-H Hariharan, A Bastianello, G Toyama, Y Shivashankar, G.V Foiani, M Sheetz, M.P DNA damage causes rapid accumulation of phosphoinositides for ATR signaling
description	10.1038/s41467-017-01805-9
author2	MECHANOBIOLOGY INSTITUTE
author_facet	MECHANOBIOLOGY INSTITUTE Wang, Y.-H Hariharan, A Bastianello, G Toyama, Y Shivashankar, G.V Foiani, M Sheetz, M.P
format	Article
author	Wang, Y.-H Hariharan, A Bastianello, G Toyama, Y Shivashankar, G.V Foiani, M Sheetz, M.P
author_sort	Wang, Y.-H
title	DNA damage causes rapid accumulation of phosphoinositides for ATR signaling
title_short	DNA damage causes rapid accumulation of phosphoinositides for ATR signaling
title_full	DNA damage causes rapid accumulation of phosphoinositides for ATR signaling
title_fullStr	DNA damage causes rapid accumulation of phosphoinositides for ATR signaling
title_full_unstemmed	DNA damage causes rapid accumulation of phosphoinositides for ATR signaling
title_sort	dna damage causes rapid accumulation of phosphoinositides for atr signaling
publisher	Nature Publishing Group
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/174372
_version_	1795374332163129344

DNA damage causes rapid accumulation of phosphoinositides for ATR signaling

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