Developmental YAPdeltaC determines adult pathology in a model of spinocerebellar ataxia type 1

10.1038/s41467-017-01790-z

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Main Authors:	Fujita, K, Mao, Y, Uchida, S, Chen, X, Shiwaku, H, Tamura, T, Ito, H, Watase, K, Homma, H, Tagawa, K, Sudol, M, Okazawa, H
Other Authors:	PHYSIOLOGY
Format:	Article
Published:	Nature Publishing Group 2020
Subjects:	ataxin 1 retinoid related orphan receptor alpha transcription factor transcription factor YAPdeltaC unclassified drug Atxn1 protein, mouse isoprotein phosphoprotein Rora protein, mouse signal transducing adaptor protein Yap protein, mouse adult cells and cell components developmental biology gene expression mutation numerical model pathology adulthood animal cell animal experiment animal model animal tissue Article controlled study functional disease genetic transcription in vivo study male mouse nonhuman protein domain protein expression protein protein interaction spinocerebellar degeneration supplementation animal cerebellum cytology disease model gene expression regulation gene knock-in genetics metabolism nerve cell pathophysiology phenotype rotarod test Mus Adaptor Proteins, Signal Transducing Animals Ataxin-1 Cerebellum Disease Models, Animal Gene Expression Regulation, Developmental Gene Knock-In Techniques Male Mice Neurons Nuclear Receptor Subfamily 1, Group F, Member 1 Phenotype Phosphoproteins Protein Isoforms Rotarod Performance Test Spinocerebellar Ataxias
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/174376
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Institution:	National University of Singapore

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spelling	sg-nus-scholar.10635-1743762024-04-04T01:11:43Z Developmental YAPdeltaC determines adult pathology in a model of spinocerebellar ataxia type 1 Fujita, K Mao, Y Uchida, S Chen, X Shiwaku, H Tamura, T Ito, H Watase, K Homma, H Tagawa, K Sudol, M Okazawa, H PHYSIOLOGY ataxin 1 retinoid related orphan receptor alpha transcription factor transcription factor YAPdeltaC unclassified drug ataxin 1 Atxn1 protein, mouse isoprotein phosphoprotein retinoid related orphan receptor alpha Rora protein, mouse signal transducing adaptor protein Yap protein, mouse adult cells and cell components developmental biology gene expression mutation numerical model pathology adulthood animal cell animal experiment animal model animal tissue Article controlled study functional disease genetic transcription in vivo study male mouse nonhuman protein domain protein expression protein protein interaction spinocerebellar degeneration supplementation animal cerebellum cytology disease model gene expression regulation gene knock-in genetics metabolism nerve cell pathophysiology phenotype rotarod test spinocerebellar degeneration Mus Adaptor Proteins, Signal Transducing Animals Ataxin-1 Cerebellum Disease Models, Animal Gene Expression Regulation, Developmental Gene Knock-In Techniques Male Mice Neurons Nuclear Receptor Subfamily 1, Group F, Member 1 Phenotype Phosphoproteins Protein Isoforms Rotarod Performance Test Spinocerebellar Ataxias 10.1038/s41467-017-01790-z Nature Communications 8 1 1864 2020-09-04T02:27:25Z 2020-09-04T02:27:25Z 2017 Article Fujita, K, Mao, Y, Uchida, S, Chen, X, Shiwaku, H, Tamura, T, Ito, H, Watase, K, Homma, H, Tagawa, K, Sudol, M, Okazawa, H (2017). Developmental YAPdeltaC determines adult pathology in a model of spinocerebellar ataxia type 1. Nature Communications 8 (1) : 1864. ScholarBank@NUS Repository. https://doi.org/10.1038/s41467-017-01790-z 2041-1723 https://scholarbank.nus.edu.sg/handle/10635/174376 Nature Publishing Group Unpaywall 20200831
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	ataxin 1 retinoid related orphan receptor alpha transcription factor transcription factor YAPdeltaC unclassified drug ataxin 1 Atxn1 protein, mouse isoprotein phosphoprotein retinoid related orphan receptor alpha Rora protein, mouse signal transducing adaptor protein Yap protein, mouse adult cells and cell components developmental biology gene expression mutation numerical model pathology adulthood animal cell animal experiment animal model animal tissue Article controlled study functional disease genetic transcription in vivo study male mouse nonhuman protein domain protein expression protein protein interaction spinocerebellar degeneration supplementation animal cerebellum cytology disease model gene expression regulation gene knock-in genetics metabolism nerve cell pathophysiology phenotype rotarod test spinocerebellar degeneration Mus Adaptor Proteins, Signal Transducing Animals Ataxin-1 Cerebellum Disease Models, Animal Gene Expression Regulation, Developmental Gene Knock-In Techniques Male Mice Neurons Nuclear Receptor Subfamily 1, Group F, Member 1 Phenotype Phosphoproteins Protein Isoforms Rotarod Performance Test Spinocerebellar Ataxias
spellingShingle	ataxin 1 retinoid related orphan receptor alpha transcription factor transcription factor YAPdeltaC unclassified drug ataxin 1 Atxn1 protein, mouse isoprotein phosphoprotein retinoid related orphan receptor alpha Rora protein, mouse signal transducing adaptor protein Yap protein, mouse adult cells and cell components developmental biology gene expression mutation numerical model pathology adulthood animal cell animal experiment animal model animal tissue Article controlled study functional disease genetic transcription in vivo study male mouse nonhuman protein domain protein expression protein protein interaction spinocerebellar degeneration supplementation animal cerebellum cytology disease model gene expression regulation gene knock-in genetics metabolism nerve cell pathophysiology phenotype rotarod test spinocerebellar degeneration Mus Adaptor Proteins, Signal Transducing Animals Ataxin-1 Cerebellum Disease Models, Animal Gene Expression Regulation, Developmental Gene Knock-In Techniques Male Mice Neurons Nuclear Receptor Subfamily 1, Group F, Member 1 Phenotype Phosphoproteins Protein Isoforms Rotarod Performance Test Spinocerebellar Ataxias Fujita, K Mao, Y Uchida, S Chen, X Shiwaku, H Tamura, T Ito, H Watase, K Homma, H Tagawa, K Sudol, M Okazawa, H Developmental YAPdeltaC determines adult pathology in a model of spinocerebellar ataxia type 1
description	10.1038/s41467-017-01790-z
author2	PHYSIOLOGY
author_facet	PHYSIOLOGY Fujita, K Mao, Y Uchida, S Chen, X Shiwaku, H Tamura, T Ito, H Watase, K Homma, H Tagawa, K Sudol, M Okazawa, H
format	Article
author	Fujita, K Mao, Y Uchida, S Chen, X Shiwaku, H Tamura, T Ito, H Watase, K Homma, H Tagawa, K Sudol, M Okazawa, H
author_sort	Fujita, K
title	Developmental YAPdeltaC determines adult pathology in a model of spinocerebellar ataxia type 1
title_short	Developmental YAPdeltaC determines adult pathology in a model of spinocerebellar ataxia type 1
title_full	Developmental YAPdeltaC determines adult pathology in a model of spinocerebellar ataxia type 1
title_fullStr	Developmental YAPdeltaC determines adult pathology in a model of spinocerebellar ataxia type 1
title_full_unstemmed	Developmental YAPdeltaC determines adult pathology in a model of spinocerebellar ataxia type 1
title_sort	developmental yapdeltac determines adult pathology in a model of spinocerebellar ataxia type 1
publisher	Nature Publishing Group
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/174376
_version_	1795374333406740480

Developmental YAPdeltaC determines adult pathology in a model of spinocerebellar ataxia type 1

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